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Possible neglected transient (T) polyagglutination in critically ill patients with coronavirus disease-2019 (Covid-19).
Pak J Pharm Sci
July 2023
Thrombosis and Vascular Diseases Laboratory, School of Health and Biomedical Sciences, Royal Melbourne Institute of Technology (RMIT) University, Bundoora, VIC, Austra.
T-activation polyagglutination can be caused by bacteria or viruses and has been associated with haemolytic anaemia. Coronavirus disease-19 (COVID-19) is also associated with haemolytic anaemia. The presented study aims to determine T activation polyagglutination in critically ill COVID-19 patients.
View Article and Find Full Text PDFPolyagglutinability phenomenon: a case report and review of the literature.
J Med Case Rep
August 2023
Laboratoire d'hématologie, CHU Mohammed VI, Faculté de Médecine et de Pharmacie d'Oujda, Université Mohammed Premier, Oujda, Morocco.
Background: Polyagglutinability of red blood cells is a rare immunological phenomenon, it is due to a cryptic antigen that is abnormally present on the surface of red blood cells. The aim of our work is to shed light on polyagglutinability, which is still poorly understood cause of discordance between the cell and serum tests and can sometimes have harmful transfusion consequences.
Case Presentation: We report the case of a 70-year-old African patient admitted for management of hemolytic anemia.
SID: a new carbohydrate blood group system based on a well-characterized but still mysterious antigen of great pathophysiologic interest.
Immunohematology
April 2023
Professor of Transfusion Medicine, Deputy Dean, Faculty of Medicine, Division of Hematology and Transfusion Medicine, Department of Laboratory Medicine, Lund University, BMC C14, Sölvegatan 19, SE-22184 Lund, Sweden, and Medical Director of the Nordic Reference Laboratory for Genomic Blood Group Typing, Department of Clinical Immunology and Transfusion Medicine, Office for Medical Services, Akutgatan 8, SE-22185, Lund, Sweden.
The high-prevalence blood group antigen, Sd, had been puzzling blood bankers and transfusionists for at least a decade when it was reported in 1967. The characteristic mix of agglutinates and free red blood cells (RBCs), caused by anti-Sd, is seen with the RBCs from 90 percent of individuals of European descent. However, only 2-4 percent of individuals are truly Sd(a-) and may produce anti-Sd.
View Article and Find Full Text PDFMinor Cross-Matching in the Diagnosis of Pneumococcal Hemolytic Uremic Syndrome in an 18-Month-Old Boy.
Lab Med
March 2023
Department of Transfusion Medicine and Blood Centre, Kalinga Institute of Medical Sciences, Bhubaneswar, India.
Article Synopsis
- Limitations in diagnostic facilities in developing nations make it challenging to identify the specific causes of hemolytic uremic syndrome (HUS).
- An 18-month-old boy in India showed severe pneumonia symptoms, abnormal lab results, and quickly deteriorated, requiring intensive care and multiple treatments.
- The case highlights the importance of minor cross-matching for diagnosing polyagglutination related to Streptococcus pneumoniae (sp-HUS), but sadly, the boy did not survive despite interventions.
A case of Tn polyagglutination discovered by an ABO blood group discrepancy.
Transfusion
July 2022
Department of Pathology and Laboratory Medicine, Center for Transfusion and Cellular Therapies, Emory University School of Medicine, Atlanta, Georgia, USA.
Background: Tn syndrome is an acquired form of polyagglutination arising from somatic mutations of hematopoietic stem cells. Tn red blood cells (RBCs) are agglutinable by naturally occurring anti-Tn antibodies in most adult sera. Current ABO typing reagents are monoclonal and do not detect polyagglutination on forward typing.
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