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Visible-light absorbing metal-free organic dyes are of increasing demand for various optoelectronic applications because of their great structure-function tunability through chemical means. Several dyes also show huge potential in triplet photosensitization, generating reactive singlet oxygen. Understanding the structure-property relationships of many well-known fluorescein dyes is of paramount importance in designing next-generation energy efficient dyes, which is currently limited.

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This paper is devoted to the investigation of the plasmonic effect of metal nanoparticles (NPs) formed on the surface of the YAG: Bi, Ce, Yb phosphors in a temperature range between 4 and 300 K. Combination of a thin conversion layer with silver plasmonic nanostructures leads to increase of sensitizer absorption and emission efficiency. Enhancement of Bi luminescence in YAG epitaxial films with Ag NPs was observed upon cooling the samples below 200 K.

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Background: Amyloid‐β (Aβ) plaques and tau pathogenesis in the brain precede cognitive decline in the progression of Alzheimer’s dementia, yet the extent to which these measures can predict localized brain tissue atrophy has not been studied in a large, diverse population. Multisite studies offer robust statistical power with larger sample sizes but are confounded by variations in biomarker quantification across studies, including variations in MRI scanners, PET tracers, and CSF assays. Longitudinal data from N=1223 individuals from four independent AD studies were harmonized to assess localized brain tissue atrophy over 2 to 5 years.

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Background: It is increasingly clear that delaying the onset of Alzheimer’s disease (AD) dementia by several years can meaningfully lower its prevalence. The goal of the present study is to examine the relationship between lifestyle activities and cognition function as well as cerebrospinal fluid (CSF) biomarkers of AD to determine whether these activities can serve as protective factors for AD resistance and resilience.

Methods: 173 cognitively normal older individuals (mean ± SD, 69 ± 6.

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Background: Mechanisms of cellular senescence affecting neuroimmune responses, homeostasis, and synaptic functioning associate with core Alzheimer’s disease (AD) biomarkers, i.e., pathological aggregation of β‐amyloid (Aβ) and tau protein, while their longitudinal associations remain unclear.

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