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Licorice Toxicity Presenting As Refractory Hypokalemia and Hypertension in an Elderly Patient.
Cureus
November 2024
Acute Medicine, Lancashire Teaching Hospitals, Lancashire, GBR.
Licorice toxicity can present with a triad of severe hypokalemia, metabolic alkalosis, and hypertension, particularly in elderly patients. We present the intriguing case of a 78-year-old male who was referred for evaluation of refractory hypokalemia and newly developed hypertension. Despite an unremarkable systemic review and minimal symptoms, a detailed dietary history revealed significant daily consumption of licorice, initially believed by the patient to support smoking cessation.
View Article and Find Full Text PDFPseudo Hyperaldosteronism Secondary to Herbal Medicine Use.
J Community Hosp Intern Med Perspect
November 2022
Maimonides Medical Center, NY, USA.
Glycyrrhizic acid, better known as licorice, is commonly found in various food and cosmetic products. Excessive consumption is known to cause a syndrome of apparent mineralocorticoid excess or pseudo hyperaldosteronism. Patients typically present with resistant hypertension and hypokalemia mimicking symptoms of primary hyperaldosteronism however laboratory workup will reveal low or normal levels of plasma renin and aldosterone in the serum.
View Article and Find Full Text PDFSyndromes of Pseudo-Hyperaldosteronism.
Clin J Am Soc Nephrol
April 2022
Department of Nephrology, University of Colorado, Aurora, Colorado.
[Hypokalemia with pseudo-hyperaldosteronism: Is it Lidl® syndrome?].
Nephrol Ther
July 2020
Division of nephrology, Rennes University Hospital, 2, rue Henri-Le-Guilloux, 35000 Rennes, France; Irset (Institut de Recherche en Santé, Environnement et Travail)-UMR 1085, 9, avenue du Professeur-Léon-Bernard, 35000 Rennes, France.
Introduction: Intoxication induced by glycyrrhizin is a common cause of hypokalaemia by pseudo-hyperaldosteronism.
Observation: We hereby present the observation of a 68-year old patient hospitalised following a full hip-prosthesis operation after a deep hypokalaemia at 2.5mM was observed, with ECG signs (flat T waves and appearance of U waves).
[Genetics and arterial hypertension: monogenic forms].
G Ital Nefrol
December 2006
Cattedra e Scuola di Specializzazione in Nefrologia, Università degli Studi di Milano, Sesto San Giovanni (MI)-Italy.
Hypertension is a complex, multifactorial disease; genetic factors represent one third to half of the inter-individual variability of blood pressure values. The study of genes involved in rare forms of monogenic hypertension led to the identification of pivotal pathophysiological pathways of kidney sodium and water reabsorption that can influence blood pressure values when changed. Glucocorticoid-Remediable Aldosteronism (GRA) is characterised by normal to high aldosterone levels, despite plasma renin activity suppression, and by the fact that these alterations are corrected by exogenous glucocorticoid administration.
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