AI Article Synopsis
- Patent foramen ovale (PFO) in carcinoid heart disease can cause hypoxia due to shunting of blood from the right to left side of the heart, worsening patient symptoms.
- Percutaneous closure of the PFO can improve oxygen saturation and alleviate related symptoms while protecting left-sided heart valves.
- A case is presented involving a 70-year-old woman with a neuroendocrine tumor who experienced improved dyspnea and oxygen levels after PFO closure using an Amplatzer occluder device.
Article Abstract
Patent foramen ovale (PFO) in patients with carcinoid heart disease (CHD) may result in hypoxia due to the presence of large right (R) to left (L) intracardiac shunts leading to hypoxia and worsening clinical condition. Percutaneous closure of the PFO can normalize oxygen saturation, relieve symptoms, and lessens left-sided heart valves involvement. We describe a case of a 70-year-old female patient with a history of small bowel neuroendocrine tumor on monthly octreotide infusion presented with worsening exertional dyspnea and hypoxia secondary to R to L intracardiac shunt through the PFO. The decision was made to close the PFO percutaneously with Amplatzer (Plymouth, MN: Abbott) PFO occluder device which resulted in immediate normalization of oxygen saturation and relief of her dyspnea.
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| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC8760016 | PMC |
| http://dx.doi.org/10.7759/cureus.20497 | DOI Listing |
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