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Long COVID-19 (LC) is a poorly understood, multifactorial condition that persists for at least three months following SARS-CoV-2 infection. The underlying pathophysiological mechanisms responsible for the wide range of associated symptoms-including fatigue, brain fog, and respiratory issues-remain unclear. However, emerging evidence suggests that the reactivation of latent viral infections, such as Epstein-Barr virus, cytomegalovirus, and varicella-zoster virus, may significantly contribute to the complexity of LC.

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Long coronavirus disease (COVID), also known as the post-acute sequelae of coronavirus disease 2019 (COVID-19) (PASC), is a significant concern since the end of the COVID-19 pandemic, as it still manifests in individuals with persistent symptoms and complications beyond the acute phase of infection. Defining this disease is challenging, as it manifests as a spectrum of symptoms varying in severity among individuals who have previously tested positive for COVID-19. Long COVID is more prevalent in hospitalized COVID-19 patients and presents in various ways, ranging from pulmonary to extrapulmonary symptoms.

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Article Synopsis
  • Sex differences in COVID-19 outcomes show that men experience greater severity and mortality during acute infection, while women are more likely to develop Long Covid (LC).
  • The study analyzed blood samples from 45 participants to explore how immune responses differ between men and women in relation to LC development, identifying sex-specific immune pathways.
  • Findings revealed that men who later developed LC had increased TGF-β signaling, while women had reduced signaling and elevated RNA associated with autoimmunity during acute infection, indicating distinct immune changes that could inform targeted treatments.
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Article Synopsis
  • - The study aimed to identify predictors of adverse outcomes in patients recovering from acute coronary syndrome (ACS) to better recognize high-risk individuals.
  • - Conducted as a retrospective cohort analysis, it followed 121 ACS patients from the University of Sarajevo over 12 months, finding that 27.3% experienced major cardiovascular events (MACE).
  • - Key predictors of MACE included older age, higher creatinine levels, lower ejection fraction, and larger left atrial diameter, highlighting the importance of careful monitoring of these factors for better patient management.
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Blood Pressure Regulation in Post-COVID POTS: Beyond Sinus Tachycardia.

Hypertension

December 2024

Department of Clinical Sciences, Lund University, Malmö, Sweden (M.J., F.R., V.H., P.M.N., A.F.).

Article Synopsis
  • Postural orthostatic tachycardia syndrome (POTS), frequently diagnosed after COVID-19, involves excessive heart rate increase when standing, without drops in blood pressure (BP). This study compared BP profiles of 100 POTS patients post-COVID-19 to 100 healthy controls.* -
  • The findings revealed that POTS patients had higher nighttime systolic BP and were more likely to experience daytime hypotensive episodes, with significant differences in circadian BP patterns.* -
  • Overall, the study suggests that individuals with post-COVID-19 POTS have disrupted BP regulation and higher average BP levels, indicating a need for further research on potential targeted BP treatments.*
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