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Correction: RANBP1 promotes colorectal cancer progression by regulating pre-miRNA nuclear export via a positive feedback loop with YAP.
Oncogene
February 2022
Department of Anatomy, Histology and Embryology, Nanjing Medical University, Nanjing, 211166, China.
Association of RANBP1 haplotype with smooth pursuit eye movement abnormality.
Am J Med Genet B Neuropsychiatr Genet
January 2011
Department of Genetic Epidemiology, SNP Genetics, Inc., Seoul, Republic of Korea.
Schizophrenia is a multifactorial disorder and smooth pursuit eye movement (SPEM) disturbance is proposed as one of the most consistent neurophysiological endophenotype in schizophrenia. The aim of this study was to examine the genetic association of RANBP1 polymorphisms with the risk of schizophrenia and with the risk of SPEM abnormality in schizophrenia patients in a Korean population. Two SNPs of RANBP1 were genotyped by TaqMan assay.
View Article and Find Full Text PDFRanGAP mediates GTP hydrolysis without an arginine finger.
Nature
February 2002
Max-Planck-Institut für molekulare Physiologie, Abteilung Strukturelle Biologie, Dortmund, Germany.
GTPase-activating proteins (GAPs) increase the rate of GTP hydrolysis on guanine nucleotide-binding proteins by many orders of magnitude. Studies with Ras and Rho have elucidated the mechanism of GAP action by showing that their catalytic machinery is both stabilized by GAP binding and complemented by the insertion of a so-called 'arginine finger' into the phosphate-binding pocket. This has been proposed as a universal mechanism for GAP-mediated GTP hydrolysis.
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