AI Article Synopsis

  • Langerhans cell histiocytosis (LCH) is a rare disease with unpredictable symptoms and limited treatment options until recent advancements.
  • The use of targeted therapies, particularly imatinib—a tyrosine kinase inhibitor—has shown promise, especially in a case with brain involvement.
  • A study evaluating both PDGFRβ expression and the clinical effects of imatinib on LCH patients found significant PDGFRβ expression in some cases and long-lasting disease control in the treated patients.

Article Abstract

Langerhans cell histiocytosis (LCH) is a rare disease that has a variable clinical presentation and unpredictable behavior. Until recently, therapeutic options were limited. Insights into the role of mitogen-activated protein kinase (MAPK) signaling have allowed the increased use of targeted treatments. Before the advent of drugs that interfere with this pathway, investigations concerning the tyrosine kinase inhibitor imatinib opened the way to a rationale-based therapeutic approach to the disease. Imatinib block the binding site of ATP in the BCR/ABL protein and is also a platelet-derived growth factor receptor (PDGFR) and a KIT (CD117) kinase inhibitor. A case of refractory LCH with brain involvement was reported to be successfully treated with imatinib. Thereafter, we further explored the role of this tyrosine kinase inhibitor. The present study is composed of an immunohistochemical evaluation of PDGFRβ expression and a clinical evaluation of imatinib in a series of LCH patients. In the first part, a series of 10 samples obtained from LCH patients was examined and a strong immunohistochemistry expression of PDGFRβ was found in 40% of the cases. In the clinical part of the study, five patients were enrolled. Long-lasting disease control was obtained. These results may suggest a potential role for this drug in the current age.

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC8698345PMC
http://dx.doi.org/10.3390/biomedicines9121759DOI Listing

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