The security of cryptocircuits is determined not only for their mathematical formulation, but for their physical implementation. The so-called fault injection attacks, where an attacker inserts faults during the operation of the cipher to obtain a malfunction to reveal secret information, pose a serious threat for security. These attacks are also used by designers as a vehicle to detect security flaws and then protect the circuits against these kinds of attacks. In this paper, two different attack methodologies are presented based on inserting faults through the clock signal or the control signal. The optimization of the attacks is evaluated under supply voltage and temperature variation, experimentally determining the feasibility through the evaluation of different Trivium versions in 90 nm ASIC technology implementations, also considering different routing alternatives. The results show that it is possible to inject effective faults with both methodologies, improving fault efficiency if the power supply voltage decreases, which requires only half the frequency of the short pulse inserted into the clock signal to obtain a fault. The clock signal modification methodology can be extended to other NLFSR-based cryptocircuits and the control signal-based methodology can be applied to both block and stream ciphers.
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http://dx.doi.org/10.3390/s21227596 | DOI Listing |
eNeuro
January 2025
University of Kassel, 34132 Kassel, Germany.
Evolutionary pressures adapted insect chemosensation to the respective insect's physiological needs and tasks in their ecological niches. Solitary nocturnal moths rely on their acute olfactory sense to find mates at night. Pheromones are detected with maximized sensitivity and high temporal resolution through mechanisms that are mostly unknown.
View Article and Find Full Text PDFPlant Cell
December 2024
Department of Plant and Microbial Biology, University of Minnesota at Twin Cities, Saint Paul, MN 55108, USA.
In Arabidopsis (Arabidopsis thaliana), light and circadian clock signaling converge on PHYTOCHROME-INTERACTING FACTORS (PIFs) 4 and 5 to produce a daily rhythm of hypocotyl elongation. PIF4 and PIF5 expression is repressed at dusk by the evening complex (EC), consisting of EARLY FLOWERING3 (ELF3), ELF4, and LUX ARRHYTHMO (LUX). Here, we report that ELF3 recruits the JUMONJI (JMJ) H3K4me3 demethylases JMJ17 and JMJ18 to the PIF4 and PIF5 loci in the evening to remove their H3K4me3 marks.
View Article and Find Full Text PDFAs a low-energy method to increase the data rate of optical links in data centers, we propose self-homodyne Nyquist optical time division multiplexing (OTDM). In Nyquist OTDM, spectrally efficient high-baud rate signals can be generated exceeding the limit of electronic signal processing. However, full integration of OTDM systems has not been reported, mainly because of the complicated signal detection scheme, which involves demultiplexing and clock recovery.
View Article and Find Full Text PDFPLoS One
January 2025
Center for Computation and Integrative Biology, Rutgers, The State of New Jersey, Camden, NJ, United States of America.
Melatonin, a molecule with diverse biological functions, is ubiquitously present in living organisms. There is significant interest in understanding melatonin signal transduction pathways in humans, particularly due to its critical role in regulating the sleep-wake cycle. However, a knowledge gap remains in fully elucidating the mechanisms by which melatonin influences circadian regulation.
View Article and Find Full Text PDFFront Neurosci
January 2025
Translational Neuroscience Program, Department of Psychiatry, University of Pittsburgh School of Medicine, Pittsburgh, PA, United States.
Introduction: Alterations in multiple subregions of the human prefrontal cortex (PFC) have been heavily implicated in psychiatric diseases. Moreover, emerging evidence suggests that circadian rhythms in gene expression are present across the brain, including in the PFC, and that these rhythms are altered in disease. However, investigation into the potential circadian mechanisms underlying these diseases in animal models must contend with the fact that the human PFC is highly evolved and specialized relative to that of rodents.
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