Here is a case of a Pulmonary AVM in a female presenting with sudden onset of dizziness and vomiting most likely secondary to a paradoxical emboli causing an ischemic stroke of the cerebellum.
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| http://dx.doi.org/10.1016/j.radcr.2021.09.029 | DOI Listing |
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Lung transplantation for diffuse pulmonary arteriovenous malformations associated with juvenile polyposis-hereditary hemorrhagic telangiectasia overlap syndrome: a case report.
Gen Thorac Cardiovasc Surg Cases
December 2024
Department of Thoracic Surgery, Kyoto University Graduate School of Medicine, Kyoto, 606-8507, Japan.
Background: Lung transplantation is a viable lifesaving option for patients with diffuse pulmonary arteriovenous malformations (AVMs). We present a case of diffuse pulmonary AVMs associated with juvenile polyposis and hereditary hemorrhagic telangiectasia (JP-HHT) that was successfully managed by lung transplantation.
Case Presentation: A 19-year-old woman developed severe hypoxemia due to pulmonary AVMs diagnosed at 4 years of age.
The "pseudo-pulmonary AVM sign": an aid to the diagnosis of histoplasmosis and differentiation from pulmonary arteriovenous malformations.
Curr Probl Diagn Radiol
December 2024
Department of Radiology, Corewell Health William Beaumont University Hospital, Royal Oak, MI, USA.
The diagnostic algorithm for histoplasmosis highlights the importance of imaging and emphasizes the role of the radiologist in the diagnostic workup. Here we describe a case series of patients with a novel sign of lung involvement in histoplasmosis which we have coined the Pseudo-Pulmonary Arteriovenous Malformation (PAVM) sign, the usage of which would help in the imaging diagnosis of histoplasmosis aid by distinguishing it from PAVMs. PAVMs carry risk for serious complications such as systemic emboli and may require treatment; whereas, histoplasmomas do not.
View Article and Find Full Text PDFUncorrectable hypoxemia due to large pulmonary arteriovenous malformation in a patient with myocardial infarction: a case report.
J Yeungnam Med Sci
December 2024
Division of Cardiology, Department of Internal Medicine, Yeungnam University Medical Center, Daegu, Korea.
Article Synopsis
- * A 78-year-old man experienced ongoing low oxygen levels after a heart attack, which was found to be due to a large pulmonary arteriovenous malformation (AVM).
- * The case highlights the need to investigate external factors like AVMs in patients with severe hypoxemia and shows how useful bedside imaging techniques can be in diagnosing these conditions.
Glenn circulation causes early and progressive shunting in a surgical model of pulmonary arteriovenous malformations.
Physiol Rep
November 2024
Department of Pediatrics, Division of Cardiology, Medical College of Wisconsin, Children's Wisconsin, Herma Heart Institute, Milwaukee, Wisconsin, USA.
Pulmonary arteriovenous malformations (PAVMs) universally develop in patients with single ventricle congenital heart disease. Single ventricle PAVMs have been recognized for over 50 years but remain poorly understood. To improve our understanding, we developed a surgical rat model of Glenn circulation and characterized PAVM physiology over multiple time points.
View Article and Find Full Text PDFCDK6-mediated endothelial cell cycle acceleration drives arteriovenous malformations in hereditary hemorrhagic telangiectasia.
Nat Cardiovasc Res
November 2024
Litwin-Zucker Alzheimer's Research Center, The Feinstein Institutes for Medical Research, Northwell Health, Manhasset, NY, USA.
Increased endothelial cell proliferation is a hallmark of arteriovenous malformations (AVMs) in hereditary hemorrhagic telangiectasia (HHT). Here, we report a cyclin-dependent kinase 6 (CDK6)-driven mechanism of cell cycle deregulation involved in endothelial cell proliferation and HHT pathology. Specifically, endothelial cells from the livers of HHT mice bypassed the G1/S checkpoint and progressed through the cell cycle at an accelerated pace.
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