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| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC8369871 | PMC |
| http://dx.doi.org/10.1007/s00063-021-00840-1 | DOI Listing |
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Lung cancer progression is characterized by intricate epigenetic changes that impact critical metabolic processes and cell death pathways. In this study, we investigate the role of histone lactylation at the AIM2 locus and its downstream effects on ferroptosis regulation and lung cancer progression. We utilized a combination of biochemical assays, including chromatin immunoprecipitation (ChIP), quantitative real-time PCR (qRT-PCR), and western blotting to assess histone lactylation levels and gene expression.
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