Air pollution increases the risk of pulmonary embolism: a meta-analysis.

Rev Environ Health

Beijing An Zhen Hospital, 2 Anzhen Rd, Chaoyang District, 100029, Chaoyang-qu, Beijing, China.

Published: June 2022

Objectives: Air pollution can lead to many cardiovascular and respiratory diseases, but the impact of air pollution on pulmonary embolism is still uncertain. We conducted a meta-analysis to assess the relationship between air pollution and pulmonary embolism.

Content: We searched PubMed, EMBASE, Web of Science, and the Cochran Library for citations on air pollutants (carbon monoxide, sulfur dioxide, nitrogen dioxide, ozone and particulate matter) and pulmonary embolism. A total of nine citations met the inclusion criteria. There is no evidence of bias. CO, SO, PM and PM had no significant effect on the occurrence of pulmonary embolism. NO and O can increase the risk of pulmonary embolism to a small extent.

Summary: This meta-analysis suggests that some air pollutants are associated with an increased risk of pulmonary embolism.

Outlook: Reducing air pollution and improving air quality can effectively reduce the risk of pulmonary embolism.

Download full-text PDF

Source
http://dx.doi.org/10.1515/reveh-2021-0035DOI Listing

Publication Analysis

Top Keywords

pulmonary embolism
24
air pollution
20
risk pulmonary
16
air
8
pulmonary
8
pollution pulmonary
8
air pollutants
8
embolism
6
pollution increases
4
risk
4

Similar Publications

This case report describes a geriatric male patient with myasthenia gravis (MG) secondary to giant thymoma, presenting with progressive muscle weakness and ptosis. The diagnosis of MG was confirmed through pathology, imaging, and laboratory evaluations. Considering the significant surgical risks associated with the giant thymoma, adjuvant chemotherapy was initiated.

View Article and Find Full Text PDF

Intravascular Ultrasound Findings in Acute and Chronic Deep Vein Thrombosis of the Lower Extremities.

Diagnostics (Basel)

February 2025

Interventional Radiology Department, AORN "A. Cardarelli", 80131 Naples, Italy.

Deep vein thrombosis (DVT) of the lower extremities, as part of venous thromboembolism disorder, is the third leading cause of acute cardiovascular syndrome after heart attack and stroke. It can result in disability due to pulmonary embolism (PE) and post-thrombotic syndrome (PTS), particularly in cases where the thrombosis extends to the iliofemoral veins. Anticoagulation therapy is effective in preventing thrombus propagation and embolism but may not be sufficient for thrombus degradation and venous patency restoration.

View Article and Find Full Text PDF

Background And Aim: Pulmonary thromboembolism (PTE) is a common cause of cardiovascular death worldwide. Due to its nonspecific clinical symptoms, PTE is easy to be missed or misdiagnosed. Pulmonary transit time (PTT) is a noninvasive cardiopulmonary hemodynamic index, which is the time required for a blood sample to pass through pulmonary circulation.

View Article and Find Full Text PDF

Background: Venous thromboembolism (VTE), including pulmonary embolism (PE) and deep vein thrombosis (DVT), is the third most common cardiovascular disease. A low amount of mitochondrial DNA copy number (mtDNA-CN) reflects mitochondrial dysfunctions and has been associations with arterial cardiovascular diseases. However, the role of mtDNA-CN in venous cardiovascular disease was unclear.

View Article and Find Full Text PDF

Want AI Summaries of new PubMed Abstracts delivered to your In-box?

Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!