ILC damage, and I'll repair it.

Immunity

Division of Immunology and Allergy, Department of Medicine Solna, Karolinska Institute and University Hospital Solna, 171 76 Stockholm, Sweden. Electronic address:

Published: June 2021

In this issue of Immunity, Wang et al. report that the recognition of lysophosphatidyl serine via the receptor GPR43 confers type 3 innate lymphoid cells with the capacity to sense damage-induced cell death, which in turn triggers interleukin-22-dependent tissue repair.

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Source
http://dx.doi.org/10.1016/j.immuni.2021.05.015DOI Listing

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