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Hyperventilation-induced drop attacks in vestibular schwanomma.
J Neurol
July 2021
Department of Neurology, Pusan National University Hospital, Pusan National University School of Medicine and Biomedical Research Institute, 179, Gudeok-ro, Seo-gu, Busan, 602-739, South Korea.
Effect of end-tidal CO2 clamping on cerebrovascular function, oxygenation, and performance during 15-km time trial cycling in severe normobaric hypoxia: the role of cerebral O2 delivery.
Physiol Rep
August 2013
Institute of Sports Sciences, Faculty of Biology and Medicine, University of Lausanne Lausanne, Switzerland ; Lemanic Doctoral School of Neuroscience, University of Lausanne Lausanne, Switzerland.
During heavy exercise, hyperventilation-induced hypocapnia leads to cerebral vasoconstriction, resulting in a reduction in cerebral blood flow (CBF). A reduction in CBF would impair cerebral O2 delivery and potentially account for reduced exercise performance in hypoxia. We tested the hypothesis that end-tidal Pco2 (PETCO2) clamping in hypoxic exercise would prevent the hypocapnia-induced reduction in CBF during heavy exercise, thus improving exercise performance.
View Article and Find Full Text PDFCentral dopamine modulates anapyrexia but not hyperventilation induced by hypoxia.
J Appl Physiol (1985)
March 2002
Departamento de Fisiologia, Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, Ribeirão Preto, SP, Brazil.
Hypoxia causes hyperventilation and decreases body temperature (T(b)) and metabolism [O(2) consumption (VO(2))]. Because dopamine (DA) is released centrally in response to peripheral chemoreceptor stimulation, we tested the hypothesis that central DA mediates the ventilatory, thermal, and metabolic responses to hypoxia. Thus we predicted that injection of haloperidol (a DA D(2)-receptor antagonist) into the third ventricle would augment hyperventilation and attenuate the drop in T(b) and VO(2) in conscious rats.
View Article and Find Full Text PDFCerebral vasoconstriction in vasovagal syncope: any link with symptoms? A transcranial Doppler study.
Circulation
November 2001
Department of Internal Medicine, Autonomic Unit, Santa Maria Nuova Hospital, Firenze, Italy.
Background: Cerebral vasoconstriction has been described previously in vasovagal syncope (VVS). This phenomenon appears paradoxical in view of the well-known decrease of systemic vascular resistances taking places during VVS. We aimed to assess (1) whether cerebral vasoconstriction in VVS is an independent paradoxical phenomenon and (2) whether cerebral vasoconstriction has any link with symptoms and/or VVS onsets.
View Article and Find Full Text PDFMiddle cerebral artery blood velocity is reduced with hyperthermia during prolonged exercise in humans.
J Physiol
July 2001
Department of Human Physiology, Institute of Exercise and Sport Sciences, University of Copenhagen, Denmark.
1. In the present study we examined the effect of hyperthermia on the middle cerebral artery mean blood velocity (MCA V(mean)) during prolonged exercise. We predicted that the cerebral circulation would be impaired when hyperthermia is present during exercise and assumed that this could be observed as a reduced MCA V(mean).
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