Background: The reduction of misconduct levels and its prevention are two important objectives in prison settings, for which several strategies and control mechanisms are implemented.
Objetive: We explore the relationship between a set of socio-demographic, judicial, affective, and personality characteristics and various expressions of aggression.
Method: An Ecuadorian sample of 675 sentenced male prisoners was assessed using the Spanish adaptation of the Personality Assessment Inventory (PAI). Three subscales of the PAI were used as outcome measures: Aggressive Attitude (AGG-A), Verbal Aggression (AGG-V), and Physical Aggression (AGG-P).
Results: Using path analysis, three models were obtained, each of which had four indicators. Borderline personality features and antisocial personality features emerged as indicators that were common to the three models. Anxiety-related disorders and non-support were indicators specific to AGG-A, whilst schizophrenia and non-support were indicators of AGG-V, and drug problems and crimes against property were indicators of AGG-P.
Conclusion: This study indicates that each expression of aggression has a different structure, which suggests its usefulness for detecting the tendency of an inmate to predominantly express one form of aggression or another.
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http://dx.doi.org/10.1016/j.jpsychires.2021.04.042 | DOI Listing |
BMC Nurs
January 2025
Department of Nursing, Faculty of Health Sciences, Gaziantep Islam Science and Technology University, Gaziantep, Turkey.
Background: Brain drain refers to the migration of qualified professionals to developed countries in search of better living and working conditions, and has become a global concern, particularly in the healthcare sector. Migration of highly skilled nurses results in increased workload for the remaining nursing staff, limited access to quality healthcare services, and contributes to disparities in healthcare. Therefore, nursing students represent a critical demographic group for understanding the drivers of brain drain in the healthcare sector.
View Article and Find Full Text PDFAnal Chim Acta
January 2025
School of Medical Technology, Guangdong Medical University, Dongguan, 523808, China; School of Basic Medical Science, Southern Medical University, Guangzhou, 510515, China. Electronic address:
Background: Extracellular Vesicles (EVs), as nano-scale vesicles rich in biological information, hold an indispensable status in the biomedical field. However, due to the intrinsic small size and low abundance of EVs, their effective detection presents significant challenges. Although various EV detection techniques exist, their sensitivity and ease of operation still need enhancement.
View Article and Find Full Text PDFCell Host Microbe
January 2025
University of Utah School of Medicine, Department of Pathology, Division of Microbiology and Immunology, Salt Lake City, UT 84211, USA. Electronic address:
Microbiota composition regulates colitis severity, yet the innate immune mechanisms that control commensal communities and prevent disease remain unclear. We show that the innate immune receptor, Clec12a, impacts colitis severity by regulating microbiota composition. Transplantation of microbiota from a Clec12a animal is sufficient to worsen colitis in wild-type mice.
View Article and Find Full Text PDFJ Psychosom Res
January 2025
Department of Consultation-Liaison-Psychiatry and Psychosomatic Medicine, University Hospital Zurich, University of Zurich, 8091 Zurich, Switzerland. Electronic address:
Objective: Previous studies implied detrimental effects of adverse childhood experiences (ACE) on cardiovascular disease and mental health. Still, data on the influence of ACE on psychological distress in patients with an implantable cardioverter-defibrillator (ICD) are lacking.
Methods: We prospectively recruited 423 patients with an ICD.
Mol Genet Metab
December 2024
Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, TX, USA; Texas Children's Hospital, Houston, TX, USA; BCM-CUHK Center of Medical Genetics, Prince of Wales Hospital, Hong Kong, China.
The 3-methylglutaconic aciduria (3-MGA-uria) syndromes comprise a heterogeneous group of inborn errors of metabolism defined biochemically by detectable elevation of 3-methylglutaconic acid (3-MGA) in the urine. In type 1 (or primary) 3-MGA-uria, distal defects in the leucine catabolism pathway directly cause this elevation. Secondary 3-MGA-uria syndromes, however, are unrelated to leucine metabolism-specific defects but share a common biochemical phenotype of elevated 3-MGA.
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