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| http://dx.doi.org/10.1016/j.jdcr.2021.01.016 | DOI Listing |
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Zinc Supplementation for Acrodermatitis Enteropathica Overlapped with Psoriasiform Lesions.
Psoriasis (Auckl)
November 2024
Department of Dermatology, Naval Medical Centre, Naval Medical University, Shanghai, 200052, People's Republic of China.
Acrodermatitis enteropathica (AE) can be caused by inherited or acquired zinc deficiency, among which site-specific skin lesions or even psoriasiform skin manifestations are present. Few cases exist in the literature involving the diagnosis and treatment of AE overlapped with psoriasiform lesions. In this case, we reported a teenage boy presented characteristic site-specific skin lesions of AE with low serum zinc level, subsequently progressed into generalized pustular psoriasiform manifestations under a genetic background, while a rapid recovery was observed after monotherapy of zinc supplementation.
View Article and Find Full Text PDFAcquired and Inherited Zinc Deficiency-Related Diseases in Children: A Case Series and a Narrative Review.
Pediatr Rep
July 2024
Pediatric Emergency Room and Emergency Medicine Unit, Emergency Department, IRCCS Istituto Giannina Gaslini, 16147 Genoa, Italy.
Article Synopsis
- - Zinc deficiency is a critical health issue for children, leading to a variety of health problems and conditions that can be either acquired or inherited.
- - This overview highlights the different symptoms, diagnostic difficulties, and treatment options for zinc deficiency in children, including issues like skin problems, slow growth, immune dysfunction, and neurological effects.
- - Addressing zinc deficiency requires collaboration among healthcare professionals such as pediatricians, dermatologists, geneticists, and nutritionists to improve care and outcomes for affected children.
Article Synopsis
- This study evaluates the characteristics and health findings of a newborn from a high-risk pregnancy with acrodermatitis enteropathica, focusing on a premature infant born at 25 weeks of gestation with low birth weight and Apgar scores.
- The main result indicates a significant deficiency of zinc, emphasizing the need to closely monitor trace elements and vitamins in such cases.
- The conclusion highlights that clinical diagnosis relies on specific symptoms and low serum zinc levels, suggesting that standard nutrient preparations are inadequate for premature infants, necessitating additional zinc supplementation for those with increased losses.
Analysis of similarities and differences between transient symptomatic zinc deficiency and acrodermatitis enteropathica in children: a case report of a Chinese Yi-ethnic infant.
BMC Pediatr
May 2024
Department of Pediatrics, Yibin Hospital Affiliated to Children's Hospital of Chongqing Medical University, No. 108, Shangmao road, Xuzhou district, Yibin, Sichuan, P.R. China.
Article Synopsis
- Transient symptomatic zinc deficiency (TSZD) is a rare but often overlooked condition primarily affecting breastfed premature infants, showing symptoms that closely resemble those of Acrodermatitis enteropathica (AE), a genetic disorder caused by mutations in the SLC39A4 gene.* -
- A case study of a 4-month-old infant diagnosed with TSZD revealed significantly low blood zinc levels, while genetic testing confirmed no mutations in the SLC39A4 gene, differentiating it from AE.* -
- The infant showed significant improvement following zinc supplementation, highlighting the importance of genetic analysis in accurately diagnosing TSZD versus AE to avoid misdiagnosis.*
Acquired diffuse palmoplantar erythema with keratoderma in Chinese patients with pustular psoriasis: A predictor for IL36 receptor antagonist c.115+6T>C mutation?
Exp Dermatol
March 2024
Department of Dermatology, National Taiwan University Hospital, Taipei, Taiwan.
Article Synopsis
- * Genetic testing for IL36RN mutations is not commonly used in diagnosing GPP, despite findings that patients with acrodermatitis continua of Hallopeau (ACH) often carry these mutations.
- * The study identified six patients with pustular psoriasis and IL36RN mutations, where five had ACH and experienced acute GPP flare-ups, suggesting that this specific symptom pattern could help predict IL36RN mutations in similar cases.
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