Background: The aims of this study were: 1) to investigate the impacts that an eccentric overload training (EOT) and a small-side game training (SSGT) have on the characteristics of the accelerations (ACC) and decelerations (DCC) of the players in a soccer match; and 2) to determine if EOT and SSGT could affect the ACC and DCC reduction over time in a soccer match.
Methods: Twenty-three female soccer players from a Spanish professional club were split into three groups: a small-sided game training group (SGG), an eccentric overload training group (EOG) and a control group (CG).
Results: The SSG improved the high intensity distance performed (ES [CI]=0.72 [0.22; 1.22]), the number of high intensity actions (ES [CI]=0.65 [0.01; 1.29]), the percentage of repeated high intensity actions (ES [CI]=0.54 [-0.17; 1.25]), the initial velocity of the ACC (ES [CI]=0.55 [-0.08; 1.17]) and the percentage of repeated accelerations (ES [CI]=0.87 [-0.18; 1.91]) with respect to the control group. The EOG obtained better results in distance travelling accelerating (ES [CI]=0.84 [0.09; 1.60]) and decelerating (ES [CI]=0.87 [0.23; 1.51]) above 3 m/s, maximum ACC (ES [CI]=1.92 [0.90; 2.94]) and DCC (ES [CI]=1.29 [0.44; 2.14]) and the average of maximum ACC (ES [CI]=0.89 [0.23; 1.54]) and DCC (ES [CI]=1.08 [0.62; 1.55]) with respect to the CG. A decrement in the ACC and DCC performance was observed between the first and last 15 minutes of the competition, except for the EOG.
Conclusions: The SSG obtained mainly improvements in variables related with efforts repetitions and the capacity of maintaining the ACC and the DCC over time, while improvements in the EOG were related to intensity in the ACC and DCC.
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http://dx.doi.org/10.23736/S0022-4707.20.11232-5 | DOI Listing |
Am J Perinatol
January 2025
Department of Pediatrics, Division of Neonatology, Oregon Health & Science University, Portland, United States.
Objective: To determine the prevalence of septo-optic-dysplasia (SOD) in patients with prenatally identified absent cavum septi pellucidi (CSP), agenesis of the corpus callosum (ACC) or dysgenesis of the corpus callosum (DCC).
Study Design: This retrospective chart review investigated neonates prenatally diagnosed with an absent CSP, ACC, or DCC who were admitted to a single quaternary academic medical center in the Pacific Northwest between 2016-2023. This prenatal diagnosis prompted a routine and protocolized postnatal workup for SOD including laboratory evaluation, imaging, and specialty consultation.
Acc Chem Res
November 2024
Department of Chemistry, Seoul National University, Seoul 08826, Republic of Korea.
ConspectusNickel pincer systems have recently attracted much attention for applications in various organometallic reactions and catalysis involving small molecule activation. Their exploration is in part motivated by the presence of nickel in natural systems for efficient catalysis. Among such systems, the nickel-containing metalloenzyme carbon monoxide dehydrogenase (CODH) efficiently and reversibly converts CO to CO at its active site.
View Article and Find Full Text PDFPrenat Diagn
September 2024
Department of Fetal Medicine and Prenatal Diagnosis Center, Shanghai First Maternity and Infant Hospital, Tongji University School of Medicine, Shanghai, China.
Objective: To assess the genetic etiologies underlying agenesis of the corpus callosum (ACC) and its pregnancy outcomes in the era of next-generation sequencing.
Methods: A retrospective analysis was conducted on prospectively collected prenatal ACC cases in which amniocentesis was performed between January 2016 and December 2022. ACC was divided into non-isolated and isolated according to the presence or absence of ultrasound abnormalities.
Cryst Growth Des
May 2024
Institute of Pharmacy, University of Innsbruck, Innrain 52c, 6020 Innsbruck, Austria.
J Clin Med
February 2024
Child Neurology and Psychiatry Unit, Department of Clinical and Experimental Medicine, University of Catania, 95123 Catania, Italy.
: Pathogenic variants in the deleted in colorectal cancer gene (DCC), encoding the Netrin-1 receptor, may lead to mirror movements (MMs) associated with agenesis/dysgenesis of the corpus callosum (ACC) and cognitive and/or neuropsychiatric issues. The clinical phenotype is related to the biological function of DCC in the corpus callosum and corticospinal tract development as Netrin-1 is implicated in the guidance of developing axons toward the midline. We report on a child with a novel inherited, monoallelic, pathogenic variant in the DCC gene.
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