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Correction to: RCC2 contributes to tumor invasion and chemoresistance to cisplatin in hepatocellular carcinoma.
Hum Cell
March 2021
Department of General Surgery, The First Hospital of Jilin University, Changchun, 130021, Jilin, China.
Coronin-1C and RCC2 guide mesenchymal migration by trafficking Rac1 and controlling GEF exposure.
J Cell Sci
October 2014
School of Biochemistry, University of Bristol, University Walk, Bristol BS8 1TD, UK
Sustained forward migration through a fibrillar extracellular matrix requires localization of protrusive signals. Contact with fibronectin at the tip of a cell protrusion activates Rac1, and for linear migration it is necessary to dampen Rac1 activity in off-axial positions and redistribute Rac1 from non-protrusive membrane to the leading edge. Here, we identify interactions between coronin-1C (Coro1C), RCC2 and Rac1 that focus active Rac1 to a single protrusion.
View Article and Find Full Text PDFTD-60 is required for interphase cell cycle progression.
Cell Cycle
March 2013
Tumor Development Program, Sanford-Burnham Medical Research Institute, La Jolla, CA, USA.
We previously identified TD-60 (RCC2) as a mitotic centromere-associated protein that is necessary for proper completion of mitosis. We now report that TD-60 is an essential regulator of cell cycle progression during interphase. siRNA suppression blocks progression of mammalian G₁/S phase cells and progression of G₂ cells into mitosis.
View Article and Find Full Text PDFAb initio chemical kinetics for the reaction of an H atom with Si3H8.
J Phys Chem A
March 2010
Center for Interdisciplinary Molecular Science, Institute of Molecular Science, National Chiao Tung University, Hsinchu 300, Taiwan.
The kinetics and mechanism for the reaction of H with Si(3)H(8) have been investigated using various theoretical methods including CCSD(T)/6-311++G(3df,2p)//B3LYP/6-311++G(3df,2p), G2M(RCC2), and CCSD(T)/6-311++G(3df,2p)//CCSD/6-311+G(d,p). The results obtained by the latter method show that H abstraction from a primary Si-H bond and a secondary Si-H bond leads to the formation of n-Si(3)H(7) and i-Si(3)H(7) products, with 3.8 (TS1) and 3.
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