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[Occupational interstitial lung diseases].
Radiologie (Heidelb)
August 2024
, Frankenstraße 24, 91096, Möhrendorf, Deutschland.
A variety of workplace exposures (organic or inorganic dusts as well as gases, fumes, or vapors) can cause diffuse interstitial lung disease. The latency period until onset of the disease can exceed 30 years. The disease course varies greatly and depends on the quantity of the inhaled substance and its fibrogenic effect.
View Article and Find Full Text PDFAluminosis pneumoconiosis presenting as hyperdense lung nodules.
Radiol Case Rep
June 2024
University of Nebraska Medical Center, Omaha, NE, USA.
We present the case of a 66-year-old man who presented with new incidentally found hyperdense pulmonary nodules. Further workup with a PET/CT revealed that the nodules were FDG-avid and that there was associated hypermetabolic lymphadenopathy. Due to his history of aluminum toxicity from welding, aluminosis pneumoconiosis was suspected.
View Article and Find Full Text PDFPathology and Mineralogy of the Pneumoconioses.
Semin Respir Crit Care Med
June 2023
Department of Pathology and Laboratory Medicine, Calgary, Alberta, Canada.
Pneumoconioses represent the spectrum of lung diseases caused by inhalation of respirable particulate matter small enough (typically <5-µm diameter) to reach the terminal airways and alveoli. Pneumoconioses primarily occur in occupational settings where workers perform demanding and skilled manual labor including mining, construction, stone fabrication, farming, plumbing, electronics manufacturing, shipyards, and more. Most pneumoconioses develop after decades of exposure, though shorter latencies can occur from more intense particulate matter exposures.
View Article and Find Full Text PDFImaging diagnosis of pneumoconiosis with predominant nodular pattern: HRCT and pathologic findings.
Clin Imaging
May 2023
Department of Environmental Medicine, Kochi Medical School, Okochokohasu, Nankoku, Kochi 783-0043, Japan. Electronic address:
The radiological patterns of known pneumoconiosis have been changing in recent years. The basic pathology in pneumoconiosis is the presence of dust macules, mixed dust fibrosis, nodules, diffuse interstitial fibrosis, and progressive massive fibrosis. These pathologic changes can coexist in dust-exposed workers.
View Article and Find Full Text PDFCritical Review on Toxicological Mechanisms Triggered by Inhalation of Alumina Nanoparticles on to the Lungs.
Biomedicines
October 2022
French Armed Forces Biomedical Research Institute (IRBA), Department of Biological Radiation Effects/Radiobiology Unit, 1 Place du Général Valérie André, BP 73, CEDEX, 91223 Brétigny-sur-Orge, France.
Alumina nanoparticles (AlO NPs) can be released in occupational environments in different contexts such as industry, defense, and aerospace. Workers can be exposed by inhalation to these NPs, for instance, through welding fumes or aerosolized propellant combustion residues. Several clinical and epidemiological studies have reported that inhalation of AlO NPs could trigger aluminosis, inflammation in the lung parenchyma, respiratory symptoms such as cough or shortness of breath, and probably long-term pulmonary fibrosis.
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