We propose a strategy with which conversational android robots can handle dialogue breakdowns. For smooth human-robot conversations, we must not only improve a robot's dialogue capability but also elicit cooperative intentions from users for avoiding and recovering from dialogue breakdowns. A cooperative intention can be encouraged if users recognize their own responsibility for breakdowns. If the robot always blames users, however, they will quickly become less cooperative and lose their motivation to continue a discussion. This paper hypothesizes that for smooth dialogues, the robot and the users must share the responsibility based on psychological reciprocity. In other words, the robot should alternately attribute the responsibility to itself and to the users. We proposed a dialogue strategy for recovering from dialogue breakdowns based on the hypothesis and experimentally verified it with an android. The experimental result shows that the proposed method made the participants aware of their share of the responsibility of the dialogue breakdowns without reducing their motivation, even though the number of dialogue breakdowns was not statistically reduced compared with a control condition. This suggests that the proposed method effectively elicited cooperative intentions from users during dialogues.
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http://dx.doi.org/10.3389/frobt.2019.00029 | DOI Listing |
Cells
December 2024
Immunology, Immunopathology and Therapeutic Chemistry, Institute of Molecular and Cellular Biology, CNRS UPR3572, 67084 Strasbourg, France.
The dialogue between T and B cells can be regulated by different mechanisms, such as co-inhibitory receptors, which therefore play a crucial role in preventing autoimmune diseases such as systemic lupus erythematosus (SLE). B and T lymphocyte attenuator (BTLA) is a co-inhibitory receptor expressed on many myeloid and lymphoid cells. Although peripheral B cells express a very high amount of BTLA, previous works in the context of autoimmunity mainly focused on T cells, and whether BTLA expression on B cells plays a role in the lupus pathogenesis is still unclear.
View Article and Find Full Text PDFPLoS Comput Biol
December 2024
Division of Biology and Biological Engineering, California Institute of Technology, Pasadena, California, United States of America.
For the vast majority of genes in sequenced genomes, there is limited understanding of how they are regulated. Without such knowledge, it is not possible to perform a quantitative theory-experiment dialogue on how such genes give rise to physiological and evolutionary adaptation. One category of high-throughput experiments used to understand the sequence-phenotype relationship of the transcriptome is massively parallel reporter assays (MPRAs).
View Article and Find Full Text PDFBurns
December 2024
Department of Clinical Medicine, University of Bergen, Bergen, Norway; Department of Thoracic Medicine, Haukeland University Hospital, Bergen, Norway; Bergen Centre for Ethics and Priority Setting, Department of Global Public Health and Primary Care, University of Bergen, Norway.
Background: Paediatric burn injuries constitute a transformative event for parents, shaping their lives in profound ways. This study explores parental experiences, coping mechanisms, and the impact of Family Burn Camp in the aftermath of paediatric burn injuries.
Methods: A qualitative study was conducted in 2019 comprising two focus group interviews with 15 parents at the Family Burn Camp in Stavern, Norway.
J Diabetes Investig
December 2024
Department of Endocrinology, Metabolism and Nephrology, Graduate School of Medicine, Nippon Medical School, Tokyo, Japan.
Objective: This ACTION-IO sub-analysis compared attitudes of people with obesity (PwO) and healthcare professionals (HCPs) between South Korea/Japan and global.
Methods: Responses from overall (body mass index [BMI] ≥ 25 kg/m) and higher BMI (30.0-34.
Life Sci
January 2025
Department of Pharmacy, Xinhua Hospital Affiliated to Shanghai Jiaotong University School of Medicine, Shanghai 200092, China. Electronic address:
Pathological vascular remodeling (VR) is characterized by structural and functional alterations in the vascular wall resulting from injury, which significantly contribute to the development of cardiovascular diseases (CVDs). The vascular wall consists primarily of endothelial cells (ECs), vascular smooth muscle cells (VSMCs), and adventitial fibroblasts (AFs), whose interactions are crucial for both the formation of the vascular system and the maintenance of mature blood vessels. Disruptions in the communication between these cell types have been implicated in the progression of VR.
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