The complete mitochondrial genome sequence of snake mackerels (Scombroidei, Gempylidae).

Mitochondrial DNA B Resour

Key Laboratory of Animal Ecology and Conservation Biology, Institute of Zoology, Chinese Academy of Sciences, Beijing, China.

Published: June 2020

For the first time, we illuminate the complete mitochondrial genome (mitogenome) sequence of the which is 16,988 bp in size and contains 13 protein-coding (PCGs), 2 rRNA genes, 22 tRNA genes, and one control region.The base composition of the mitogenome is 26.08% A, 26.77% T, 28.46% C and 18.69% G. Here, we selected 11 genera of species from the mostly monotypic snake mackerel family, including representative Antarctic that have been identified, and constructed phylogenetic trees to better study the snake mackerel family.

Download full-text PDF

Source
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC7782299PMC
http://dx.doi.org/10.1080/23802359.2020.1775506DOI Listing

Publication Analysis

Top Keywords

complete mitochondrial
8
mitochondrial genome
8
snake mackerel
8
mackerel family
8
genome sequence
4
sequence snake
4
snake mackerels
4
mackerels scombroidei
4
scombroidei gempylidae
4
gempylidae time
4

Similar Publications

Background: Tea-oil Camellia within the genus Camellia is renowned for its premium Camellia oil, often described as "Oriental olive oil". So far, only one partial mitochondrial genomes of Tea-oil Camellia have been published (no main Tea-oil Camellia cultivars), and comparative mitochondrial genomic studies of Camellia remain limited.

Results: In this study, we first reconstructed the entire mitochondrial genome of C.

View Article and Find Full Text PDF

Basic Science and Pathogenesis.

Alzheimers Dement

December 2024

Centre for Brain Research, Indian Institute of Science, Bengaluru, Karnataka, India.

Background: Vascular Dementia (VaD) is the second most prevalent cause of dementia, arising from the blockage of blood vessels in the brain. One event responsible for the blockage or narrowing of small blood vessels is transient ischemic attack (TIA), and these changes resolve within 24 hours in humans. The molecular mechanism underlying these changes in recovery in small vessels still needs to be investigated.

View Article and Find Full Text PDF

Background: The medical and social history of patients with Alzheimer's Disease is heterogeneous with many interacting genetic and environmental factors contributing to an individual's risk. Moreover, a maternal family history (mFH) is a key risk factor for AD-raising the risk for disease onset by as much as nine times. However, a proportion of individuals do not have a complete knowledge of their family history.

View Article and Find Full Text PDF

The complete mitochondrial genome of the deep-sea methanotrophic sponges and : leveraging 'waste' in metagenomic data.

J Genet

January 2025

Programa de Pos-graduacao em Ciencias Biologicas (Genetica), Universidade Federal do Rio de Janeiro, Rio de Janeiro, Brazil.

A significant proportion of next-generation sequencing (NGS) data ends up not being used since they comprise information out-of-scope of the primary studies. This 'waste' of potential can be harnessed to explore organellar genomes, such as the mitochondrial DNA, and be used for evolutionary, conservation and biodiversity research. We present the complete mitochondrial genomes of the deep-sea methanotrophic sponges and (Demospongiae, Poecilosclerida) retrieved from previously published whole metagenome sequencing data.

View Article and Find Full Text PDF

From Ca dysregulation to heart failure: β-adrenoceptor activation by RKIP postpones molecular damages and subsequent cardiac dysfunction in mice carrying mutant PLN by correction of aberrant Ca-handling.

Pharmacol Res

December 2024

Institute of Pharmacology and Toxicology, University of Würzburg, Versbacher Str. 9, Würzburg 97078, Germany; Leibniz-Institut für Analytische Wissenschaften - ISAS - e.V., Bunsen-Kirchhoff-Str. 11, Dortmund 44139, Germany; Comprehensive Heart Failure Center, University Hospital of Würzburg, Am Schwarzenberg 15, Würzburg 97078, Germany. Electronic address:

Article Synopsis
  • Impaired calcium (Ca) handling in heart cells is a key feature of heart failure (HF), leading to issues like weakened heart contractions and irregular heartbeats.
  • The study used transgenic mice with a mutation affecting a calcium regulator (phospholamban) to understand how defects in calcium cycling contribute to HF, noting that these mice experience severe and fast-progressing heart failure.
  • Early treatment aimed at correcting calcium cycling using Raf kinase inhibitor protein (RKIP) was found to delay heart cell damage and improve overall health of the mice, indicating that addressing Ca dynamics early on could be crucial for preventing further complications in heart failure.
View Article and Find Full Text PDF

Want AI Summaries of new PubMed Abstracts delivered to your In-box?

Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!