Calcific tendinitis is the leading cause of shoulder pain. Among patients with calcific tendinitis, 2.7%-20% are asymptomatic, and 35%-45% of patients whose calcific deposits are inadvertently discovered develop shoulder pain. If symptoms are present, complications such as decreased range of motion of the shoulder joint should be minimized while managing pain. Patients with acute calcific tendinitis respond well to conservative treatment and rarely require surgery. In contrast, patients with chronic calcific tendinitis often do not respond to conservative treatment and do require surgery. Clinical improvement takes time, even after surgical treatment. This review article summarizes the processes related to the diagnosis and treatment of calcific tendinitis with the aim of helping clinicians choose appropriate treatment options for their patients.
Download full-text PDF |
Source |
---|---|
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC7726362 | PMC |
http://dx.doi.org/10.5397/cise.2020.00318 | DOI Listing |
J Coll Physicians Surg Pak
January 2025
Orthopaedics Department, Gansu Provincial Hospital, Gansu, China.
Null.
View Article and Find Full Text PDFAm J Physiol Endocrinol Metab
January 2025
Department of Cardiology, Zhongda Hospital, School of Medicine, Southeast University 87 Dingjiaqiao, Nanjing, P.R. China.
Autophagic flux blockade and excessive oxidative stress play important roles in the pathogenesis of diabetic vascular calcification (VC). Transcription factor EB (TFEB) is an important regulator of many autophagy-lysosomal related components, which is mainly involved in promoting autophagy process in cells. Nuclear factor erythroid-2 related factor 2 (Nrf2) antioxidant system is considered as one of the key pathways in response to intracellular oxidative stress.
View Article and Find Full Text PDFJ Orthop
July 2025
Department of Surgery and Perioperative Care, Dell Medical School-The University of Texas at Austin, Austin, TX, USA.
Background: The pathophysiology of enthesopathy and tendinopathy is mucoid degeneration, which includes chondroid metaplasia. The chondroid metaplasia can be associated with calcification. Inflammation is typically absent unless calcification triggers a self-limited immune response representing acute calcific tendinitis.
View Article and Find Full Text PDFJ Orthop Case Rep
January 2025
Department of Orthopaedics, Royal Free Hospital, University College London Royal Free Hospital, London, England.
Introduction: Painter first described painful periarticular soft-tissue calcium deposits in 1907. Further research has led to a variety of nomenclature, including calcareous tendinitis, pseudopodagra, and rheumatism. This report details the journey of a patient with acute calcific periarthritis (ACP) and explores issues concerning diagnosis, management, and provides possible preventative strategies.
View Article and Find Full Text PDFJ Orthop Case Rep
January 2025
Department of Orthopaedics, ACS Medical College and Hospital, Dr. MGR Educational and Research Institute, Chennai, Tamil Nadu, India.
Introduction: Calcific tendonitis is characterized by calcium hydroxyapatite crystal deposition in tendons, leading to inflammation and pain. While predominantly observed in the rotator cuff tendons of the shoulder, its occurrence in the rectus femoris tendon of the hip is exceedingly rare and poses a diagnostic challenge.
Case Report: A 38-year-old female housewife presented with a 1-month history of left hip pain, which was dull, aching, and exacerbated by movements such as standing and walking.
Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!