We hypothesized that because Covid-19 (C19) remains an urgent and visible threat, efforts to combat its negative health consequences have become moralized. This moralization of health-based efforts may generate asymmetries in judgement, whereby harmful by-products of those efforts (i.e., instrumental harm) are perceived as more acceptable than harm resulting from non-C19 efforts, such as prioritizing the economy or non-C19 issues. We tested our predictions in two experimental studies. In Study 1, American participants evaluated the same costs (public shaming, deaths and illnesses, and police abuse of power) as more acceptable when they resulted from efforts to minimize C19's health impacts, than when they resulted from non-health C19 efforts (e.g., prioritizing economic costs) or efforts unrelated to C19 (e.g., reducing traffic deaths). In Study 2, New Zealand participants less favorably evaluated the quality of a research proposal empirically questioning a C19 elimination strategy in NZ than one questioning strategy, although both proposals contained the same amount of methodology information. This finding suggests questioning elimination approaches is morally condemned, a similar response to that found when sacred values are questioned. In both studies, condition effects were mediated by lowered moral outrage in response to costs resulting from pursuing health-minded C19 efforts. Follow-up analyses revealed that both heightened personal concern over contracting C19 and liberal ideology were associated with greater asymmetries in human cost evaluation. Altogether, results suggest efforts to reduce or eliminate C19 have become moralized, generating asymmetries in evaluations of human suffering.
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http://dx.doi.org/10.1016/j.jesp.2020.104084 | DOI Listing |
Sci Rep
December 2024
India Meteorological Department, New Delhi, 110003, India.
Desert locusts, notorious for their ruinous impact on agriculture, threaten over 20% of Earth's landmass, prompting billions in losses and global food scarcity concerns. With billions of these locusts invading agrarian lands, this is no longer a thing of the past. Recent invasions, such as those in India, where losses reached US$ 3 billion in 2019-20 alone, underscore the urgency of action.
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December 2024
Department of Ecology, Faculty of Sciences, Institute of Biology, University of Pécs, Ifjúság útja 6, Pécs, 7624, Hungary.
The European pond turtle (Emys orbicularis) is a wide-ranging, long-living freshwater species with low reproductive success, mainly due to high predation pressure. We studied how habitat variables and predator communities in near-natural marshes affect the survival of turtle eggs and hatchlings. We followed the survival of artificial turtle nests placed in marshes along Lake Balaton (Hungary) in May and June as well as hatchlings (dummies) exposed in September.
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December 2024
Department of Mathematics, Texas A&M University, College Station, TX, 77843, USA.
The northern Gulf of Mexico (nGoM) receives water from over 50 rivers which are highly influenced by humans and include the largest river in the United States, the Mississippi River. To support large-scale data-driven research centered on the dynamic river-ocean system in the region, this study consolidated hydrogeochemical river and ocean data from across the nGoM. In particular, we harmonized 35 chemical solute parameters from 54 rivers and incorporated river discharge data to derive daily solute concentration and flux estimates throughout the nGoM.
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December 2024
Department of Genetics, Yale University, Yale School of Medicine, New Haven, 06510, CT, USA.
The cis-regulatory elements encoded in an mRNA determine its stability and translational output. While there has been a considerable effort to understand the factors driving mRNA stability, the regulatory frameworks governing translational control remain more elusive. We have developed a novel massively parallel reporter assay (MPRA) to measure mRNA translation, named Nascent Peptide Translating Ribosome Affinity Purification (NaP-TRAP).
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December 2024
Department of Theory and Bio-Systems, Max Planck Institute of Colloids and Interfaces, 14476, Potsdam, Germany.
Neurodegeneration in Huntington's disease (HD) is accompanied by the aggregation of fragments of the mutant huntingtin protein, a biomarker of disease progression. A particular pathogenic role has been attributed to the aggregation-prone huntingtin exon 1 (HTTex1), generated by aberrant splicing or proteolysis, and containing the expanded polyglutamine (polyQ) segment. Unlike amyloid fibrils from Parkinson's and Alzheimer's diseases, the atomic-level structure of HTTex1 fibrils has remained unknown, limiting diagnostic and treatment efforts.
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