Power-law distributed cascade failures are well known in power-grid systems. Understanding this phenomena has been done by various DC threshold models, self-tuned at their critical point. Here, we attempt to describe it using an AC threshold model, with a second-order Kuramoto type equation of motion of the power-flow. We have focused on the exploration of network heterogeneity effects, starting from homogeneous two-dimensional (2D) square lattices to the US power-grid, possessing identical nodes and links, to a realistic electric power-grid obtained from the Hungarian electrical database. The last one exhibits node dependent parameters, topologically marginally on the verge of robust networks. We show that too weak quenched heterogeneity, coming solely from the probabilistic self-frequencies of nodes (2D square lattice), is not sufficient for finding power-law distributed cascades. On the other hand, too strong heterogeneity destroys the synchronization of the system. We found agreement with the empirically observed power-law failure size distributions on the US grid, as well as on the Hungarian networks near the synchronization transition point. We have also investigated the consequence of replacing the usual Gaussian self-frequencies to exponential distributed ones, describing renewable energy sources. We found a drop in the steady state synchronization averages, but the cascade size distribution, both for the US and Hungarian systems, remained insensitive and have kept the universal tails, being characterized by the exponent τ ≃ 1.8 . We have also investigated the effect of an instantaneous feedback mechanism in case of the Hungarian power-grid.

Download full-text PDF

Source
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC7517205PMC
http://dx.doi.org/10.3390/e22060666DOI Listing

Publication Analysis

Top Keywords

cascade failures
8
power-law distributed
8
power-grid
5
power-law
4
power-law distributions
4
distributions dynamic
4
dynamic cascade
4
failures power-grid
4
power-grid models
4
models power-law
4

Similar Publications

Prostate cancer remains a significant global health concern, with over 1.4 million new cases diagnosed and more than 330,000 deaths each year. The primary clinical challenge that contributes to poor patient outcomes involves the failure to accurately predict and treat at the onset of metastasis, which remains an incurable stage of the disease.

View Article and Find Full Text PDF

Unlabelled: Persistent viral infections can be an important medical problem, with persistently infected (PI) cells extending viral shedding, maintaining inflammation, and providing potential sources for new viral variants. Given that PI cells can acquire resistance to some innate immune pathways, we tested the hypothesis that complement (C')-mediated lysis of parainfluenza virus 5 (PIV5)-infected cells would differ between acute-infected and PI cells. Biochemical and real-time cell viability assays showed effective C'-mediated lysis of A549 lung cells acutely infected with PIV5, through pathways that depended on C3 and C5, but largely independent of C6.

View Article and Find Full Text PDF

Development of specific therapies addressing the underlying diseases' mechanisms constitutes the basis of precision medicine. Transthyretin cardiac amyloidosis (ATTR-CM) is an exemplar of precise therapeutic approach in the field of heart failure and cardiomyopathies. A better understanding of the underlying pathophysiology, more precise data of its epidemiology, and advances in imaging techniques that allow non-invasive diagnosis have fostered the development of new and very effective specific therapies for ATTR-CM.

View Article and Find Full Text PDF

Human adenovirus is an infectious agent that causes respiratory infections in adults and children. It has been found that immunocompromised children are highly susceptible to this pathogen, as it can swiftly evolve into severe pneumonia with multiple sequelae. Due to the lack of immunity in children, the body's response mechanisms to innate and acquired immunity are specialized.

View Article and Find Full Text PDF

Heart failure with preserved ejection fraction (HFpEF) has recently emerged as an insidiously and increasingly prevalent heart failure phenotype. HFpEF often occurs in the context of hypertension and obesity and presents with diastolic dysfunction, ventricular hypertrophy, and myocardial fibrosis. Despite growing study of HFpEF, the causal links between early metabolic changes, bioenergetic perturbations, and cardiac structural remodeling remain unclear.

View Article and Find Full Text PDF

Want AI Summaries of new PubMed Abstracts delivered to your In-box?

Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!