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Adopting GPR75 in treating obesity: unraveling the knowns and unknowns of this orphan GPCR.
Trends Cell Biol
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Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390, USA; Division of Endocrinology, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX 75390, USA. Electronic address:
G protein-coupled receptor 75 (GPR75) is emerging as a promising target for obesity treatment, but its exact role in energy regulation remains unclear. This article explores the latest research on GPR75's molecular function, potential ligands, and therapeutic challenges in addressing obesity.
View Article and Find Full Text PDFUnraveling the Causal Relationship Between Non-Communicable Diseases, Obesity, and Health Expenditure: Insights from the Toda-Yamamoto Approach.
Healthcare (Basel)
December 2024
Faculty of Health Sciences, Department of Nutrition and Dietetics, Haliç University, 34060 Istanbul, Turkey.
Background/objectives: Understanding the relationship between non-communicable diseases (NCDs), obesity, and health expenditure is crucial for developing effective public health policies, particularly in light of the rising global burden of NCDs and obesity. Therefore, this study aimed to investigate the causal relationships between NCDs, obesity, and health expenditure in Turkiye.
Methods: Data were collected from the World Health Organization and Our World in Data.
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Food is Medicine Institute, Friedman School of Nutrition Science and Policy, and Tufts University School of Medicine, Tufts University, Boston, MA.
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School of Psychology, Laval University, 2325 Rue des Bibliothèques, Quebec City, QC G1V 0A6, Canada.
: Binge eating (BE) is associated with physical and psychological consequences, such as obesity and reduced quality of life. The relationship between binge eating and childhood experiences of interpersonal trauma has been explored, yet few studies focus on the processes that may explain this association. In this regard, some personality traits and maladaptive cognitive-emotional regulation may help explain this relationship, as they have been associated, respectively, with BE and childhood interpersonal trauma.
View Article and Find Full Text PDFMetabolic Syndrome, Kidney-Related Adiposity, and Kidney Microcirculation: Unraveling the Damage.
Biomedicines
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Division of Nephrology and Research Institute for Convergence of Biomedical Science and Technology, Pusan National University Yangsan Hospital, Yangsan 50612, Republic of Korea.
Metabolic syndrome (MetS) is a cluster of interrelated risk factors, including insulin resistance, hypertension, dyslipidemia, and visceral adiposity, all of which contribute to kidney microvascular injury and the progression of chronic kidney disease (CKD). However, the specific impact of each component of MetS on kidney microcirculation remains unclear. Given the increasing prevalence of obesity, understanding how visceral fat-particularly fat surrounding the kidneys-affects kidney microcirculation is critical.
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