An amendment to this paper has been published and can be accessed via the original article.
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http://dx.doi.org/10.1186/s12913-020-05519-5 | DOI Listing |
J Orthop Translat
November 2024
Department of Orthopedics, The First Affiliated Hospital of Third Military Medical University (Army Medical University), Chongqing, PR China.
J Orthop Translat
November 2024
Department of Orthopaedics, The Third Xiangya Hospital, Central South University, Changsha, 410013, China.
[This corrects the article DOI: 10.1016/j.jot.
View Article and Find Full Text PDFClin Epigenetics
December 2024
The State Key Laboratory of Bioactive Substance and Function of Natural Medicines, Beijing, China.
Sci Rep
December 2024
School of Human Sciences, University of Western Australia, Crawley, WA, 6009, Australia.
Hearing loss (HL) in mid-life has been suggested as a risk factor for cognitive decline. It is unclear whether this relationship is due to deprivation of auditory input alone, degenerative processes, or a combination. Animal models are useful to investigate underlying neural mechanisms as human studies can be confounded by various factors.
View Article and Find Full Text PDFSci Rep
December 2024
Department of Physiology, School of Medicine, University of Louisville, Louisville, KY, USA.
Background -Smoking is associated with arrhythmia and sudden cardiac death, but the biological mechanisms remain unclear. In electrocardiogram (ECG) recordings abnormal durations of ventricular repolarization (QT interval), atrial depolarization (P wave), and atrioventricular depolarization (PR interval and segment), predict cardiac arrhythmia and mortality. Previous analyses of the National Health and Nutrition Examination Survey (NHANES) database for associations between smoking and ECG abnormalities were incomplete.
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