Estimated 24-Hour Urinary Sodium Excretion and Incident Cardiovascular Disease and Mortality Among 398 628 Individuals in UK Biobank.

Hypertension

From the Department of Epidemiology and Biostatistics, School of Public Health (P.E., D.C.M., D.S.-L., R.P., E.E., A.D., B.N., I.T.), Imperial College London, United Kingdom.

Published: September 2020

AI Article Synopsis

  • The study analyzed the relationship between 24-hour urinary sodium excretion (used as a proxy for sodium intake) and cardiovascular disease (CVD) and mortality among nearly 400,000 UK Biobank participants aged 40-69 with no prior health issues.
  • The analysis revealed no significant connection between sodium excretion and overall CVD, coronary heart disease, or stroke, though the results showed a suggested inverse link with heart failure that was inconclusive upon further analysis.
  • Interestingly, a J-shaped association was found between sodium excretion and mortality rates, indicating that while sodium intake may not correlate with CVD risk, it does relate to overall mortality across various diseases, but the reasons for these findings remain unclear.

Article Abstract

We report on an analysis to explore the association between estimated 24-hour urinary sodium excretion (surrogate for sodium intake) and incident cardiovascular disease (CVD) and mortality. Data were obtained from 398 628 UK Biobank prospective cohort study participants (40-69 years) recruited between 2006 and 2010, with no history of CVD, renal disease, diabetes mellitus or cancer, and cardiovascular events and mortality recorded during follow-up. Hazard ratios between 24-hour sodium excretion were estimated from spot urinary sodium concentrations across incident CVD and its components and all-cause and cause-specific mortality. In restricted cubic splines analyses, there was little evidence for an association between estimated 24-hour sodium excretion and CVD, coronary heart disease, or stroke; hazard ratios for CVD (95% CIs) for the 15th and 85th percentiles (2.5 and 4.2 g/day, respectively) compared with the 50th percentile of estimated sodium excretion (3.2 g/day) were 1.05 (1.01-1.10) and 0.96 (0.92-1.00), respectively. An inverse association was observed with heart failure, but that was no longer apparent in sensitivity analysis. A J-shaped association was observed between estimated sodium excretion and mortality. Our findings do not support a J-shaped association of estimated sodium excretion with CVD, although such an association was apparent for all-cause and cause-specific mortality across a wide range of diseases. Reasons for these differences are unclear; methodological limitations, including the use of estimating equations based on spot urinary data, need to be considered in interpreting our findings.

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http://dx.doi.org/10.1161/HYPERTENSIONAHA.119.14302DOI Listing

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