Background: Limited work has been done to explain how work-family conflict is related to anxiety symptoms and the roles of emotional exhaustion and social support may play.
Methods: Based on a sample of 764 female nurses and physicians, a model was tested in which emotional exhaustion served as a mediator and social support was regarded as a moderator between work-family conflict and anxiety symptoms.
Results: This current study supported a moderated mediation model where the relationship between work-family conflict and anxiety symptoms via emotional exhaustion was weakest for female medical staff who reported high levels of social support.
Conclusions: This study contribute to providing an understanding of how and when work-family conflict affects anxiety symptoms. The results implicate a wide range of interventions aimed at promoting mental wellbeing among female medical staff for policymakers and individuals.
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http://dx.doi.org/10.1186/s12888-020-02673-2 | DOI Listing |
Front Psychol
January 2025
KBR, Behavioral Health and Performance Laboratory, Biomedical Research and Environmental Sciences Division, Human Health and Performance Directorate, NASA Johnson Space Center, Houston, TX, United States.
While there is a large body of research on social support in traditional work settings, less is known about how the unique context of long-term isolation and confinement affects perceived social support. The purpose of our research was to examine how perceptions of social support change over time, how they differ by source (i.e.
View Article and Find Full Text PDFJ Imaging Inform Med
January 2025
Department of Radiology, Mayo Clinic, Rochester, MN, USA.
Exam protocoling is a significant non-interpretive task burden for radiologists. The purpose of this work was to develop a natural language processing (NLP) artificial intelligence (AI) solution for automated protocoling of standard abdomen and pelvic magnetic resonance imaging (MRI) exams from basic associated order information and patient metadata. This Institutional Review Board exempt retrospective study used de-identified metadata from consecutive adult abdominal and pelvic MRI scans performed at our institution spanning 2.
View Article and Find Full Text PDFRadiography (Lond)
January 2025
City St Georges, University of London, Northampton Square, London EC1V 0HB, United Kingdom.
Introduction: The demand for diagnostic imaging in emergency clinical settings has increased in recent years, placing pressure on the available workforce. However, challenges including staff shortages and inflexible shift patterns may prompt radiographers to leave emergency settings. This study explores the lived experiences of radiographers who have transitioned to non-emergency settings and factors that might influence their decision to return.
View Article and Find Full Text PDFMol Biol Rep
January 2025
Department of Pharmaceutical Control, School of Pharmacy, Mashhad University of Medical Sciences, Mashhad, Iran.
Background: Pseudomonas aeruginosa's inherent and adapted resistance makes this pathogen a serious problem for antimicrobial treatments. Furthermore, its biofilm formation ability is the most critical armor against antimicrobial therapy, and the virulence factors, on the other hand, contribute to fatal infection and other recalcitrant phenotypic characteristics. These capabilities are harmonized through cell-cell communication called Quorum Sensing (QS), which results in gene expression regulation via three major interconnected circuits: las, rhl, and pqs system.
View Article and Find Full Text PDFOnco Targets Ther
January 2025
Department of Pharmacology, adMare BioInnovations, Montréal, Quebec, H4S 1Z9, Canada.
The gene is nearly ubiquitously subjected to activating mutation in pancreatic adenocarcinomas (PDAC), occurring at a frequency of over 90% in tumors. Mutant KRAS drives sustained signaling through the MAPK pathway to affect frequently disrupted cancer phenotypes including transcription, proliferation and cell survival. Recent research has shown that PDAC tumor growth and survival required a guanine nucleotide exchange factor for RAS homolog family member A (RhoA) called GEF-H1.
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