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Expedient assessment of post-infarct remodeling by native cardiac magnetic resonance imaging in mice.
Sci Rep
June 2021
Department of Cardiac Surgery, Rostock University Medical Center, Rostock, Germany.
Novel therapeutic strategies aiming at improving the healing process after an acute myocardial infarction are currently under intense investigation. The mouse model plays a central role for deciphering the underlying mechanisms on a molecular and cellular level. Therefore, we intended to assess in-vivo post-infarct remodeling as comprehensively as possible using an expedient native magnetic resonance imaging (MRI) in the two most prominent infarct models, permanent ligation (PL) of the left anterior descending artery (LAD) versus ischemia reperfusion (I/R).
View Article and Find Full Text PDFCorrection to: Deciphering post-infarct inflammation: Should it heal, would it hurt?
J Nucl Cardiol
August 2020
Mount Sinai Morningside Hospital, Icahn School of Medicine at Mount Sinai, 421 W. 113th Street, Suite 130, New York, NY, 10025, USA.
In the original version of the editorial, a wrong figure was used. The original article has been corrected.
View Article and Find Full Text PDFDeciphering post-infarct inflammation: Should it heal, would it hurt?
J Nucl Cardiol
December 2020
Mount Sinai Morningside Hospital, Icahn School of Medicine at Mount Sinai, 421 W. 113th Street, Suite 130, New York, NY, 10025, USA.
Unraveling the Molecular Mechanism of Action of Empagliflozin in Heart Failure With Reduced Ejection Fraction With or Without Diabetes.
JACC Basic Transl Sci
November 2019
Heart Institute, Hospital Universitari Germans Trias i Pujol, Badalona, Spain.
The mechanism of action of empagliflozin in heart failure with reduced ejection fraction (HFrEF) was deciphered using deep learning in silico analyses together with in vivo validation. The most robust mechanism of action involved the sodium-hydrogen exchanger (NHE)-1 co-transporter with 94.7% accuracy, which was similar for diabetics and nondiabetics.
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