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http://dx.doi.org/10.5152/tjg.2019.18832 | DOI Listing |
Medicine (Baltimore)
December 2024
Department of Gastroenterology, The Second Affiliated Hospital of Nanchang University, Jiangxi, China.
Rationale: Pancreaticopleural fistula (PPF) is an infrequent etiology of pleural effusion, characterized by nonspecific thoracic symptoms, which often leads to misdiagnosis and subsequent severe complications. Consequently, early diagnosis is crucial for effective management and the prevention of adverse outcomes. This report presents a rare case of PPF causing bilateral pleural effusions, aiming to enhance clinical recognition of this condition.
View Article and Find Full Text PDFTurk J Surg
June 2024
Department of Hepato-Pancreato Biliary Surgery and Liver Transplant, Mahatma Gandhi University of Medical Sciences, Jaipur, India.
Clin J Gastroenterol
November 2024
Royal North Shore Hospital, Reserve Road, St Leonards, NSW, 2065, Australia.
J Endocr Soc
October 2024
Molecular and Translational Pharmacology, Department of Biomedical Sciences, University of Copenhagen, DK 2200 Copenhagen, Denmark.
Context: Excessive eating and intake of a Western diet negatively affect the intestinal immune system, resulting in compromised glucose homeostasis and lower gut bacterial diversity. The G protein-coupled receptor GPR183 regulates immune cell migration and intestinal immune response and has been associated with tuberculosis, type 1 diabetes, and inflammatory bowel diseases.
Objective: We hypothesized that with these implications, GPR183 has an important immunometabolic role and investigated this using a global Gpr183 knockout mouse model.
Rev Med Inst Mex Seguro Soc
January 2024
Universidad de Monterrey, Escuela de Medicina, Departamento Académico. Monterrey, Nuevo León, México.
Chronic alcohol consumption that begins during adolescence produces a deleterious effect on different organs, liver, skin, lung, pancreas, brain, among others. At the lung level, alcohol metabolites specifically affect apical cilia, type II alveolar epithelial cells, macrophages, and the blood-air membrane. Constituting the alcoholic lung phenotype, which increases the risk of developing lung infections, direct damage, exacerbated symptoms and causes an increase in mortality in many other lung diseases.
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