Following publication of the original article [1], the authors noticed that the following errors were introduced by pdf/html formatting issues.
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| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC6794891 | PMC |
| http://dx.doi.org/10.1186/s12859-019-3102-0 | DOI Listing |
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Background: Autism spectrum disorder (ASD) has been reported to confer an increased risk of natural premature death. Telomere erosion caused by oxidative stress is a common consequence in age-related diseases. However, whether telomere length (TL) and oxidative indicators are significantly changed in ASD patients compared with controls remains controversial.
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