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Background: An Army Reserve Combat Medic's training is focused on knowledge attainment, skill development, and building experience and training to prepare them to perform in austere conditions with limited resources like on the battlefield. Unfortunately, the exposure to skills they may be responsible for performing is limited. Research shows that greater than 90% of battlefield deaths occur in the prehospital setting, 24% of which are potentially survivable.
View Article and Find Full Text PDFA medic's responsibility: a report from the Paris refugee camps.
Lancet
September 2019
Medical Aid for Refugees and Displaced People, King's College Hospital, London, UK.
Mechanisms contributing to differential regulation of PAX3 downstream target genes in normal human epidermal melanocytes versus melanoma cells.
PLoS One
April 2016
School of Medical Sciences, Edith Cowan University, Perth, Australia; Curtin Health Innovation Research Institute of Ageing and Chronic Disease, Curtin University, Perth, Australia.
Melanoma is a highly aggressive and drug resistant form of skin cancer. It arises from melanocytes, the pigment producing cells of the skin. The formation of these melanocytes is driven by the transcription factor PAX3 early during embryonic development.
View Article and Find Full Text PDF[Significance of merosin and sarcoglycan in manifestations of certain forms of muscular dystrophy].
Srp Arh Celok Lek
April 1998
Department of Neurology, Dr. Dragisha Mishovitch Hospital-Clinical Centre, Belgrade.
The muscular dystrophy is a progressive genetically determined disease of skeletal muscles. Thanks to the development of molecular genetics there have been identified genes responsible for synthesis of the corresponding proteins of the muscular membrane, whose deficiency causes certain types of muscular dystrophy. Thus, recently has been reported that deficiency of sarcoglycan (alpha, beta, gamma and sigma) is responsible for the appearance of four different types of autosomal recessive limb-girdle muscular dystrophies (2D, 2E, 2C, 2F), and that the lack of merosin is the cause of serious merosin deficiency, of congenital muscular dystrophy.
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