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Role of Long Intergenic Nonprotein-Coding RNA 00511 in Nod-Like Receptor Protein Pyrin Domain 3-Induced Chondrocyte Pyroptosis via the MicroRNA-9-5p/FUT1 Axis.
J Microbiol Biotechnol
July 2024
School of Rehabilitation Science, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, P.R. China.
This study aimed to determine the function of LINC00511 in Nod-Like Receptor Pyrin Domain 3 inflammasome-mediated chondrocyte pyroptosis via the regulation of miR-9-5p and FUT 1. Chondrocyte inflammatory injury was induced by treating chondrocytes with LPS. Afterwards, the levels of IL-1β and IL-18, the expression of NLRP3, ASC, Caspase-1, and GSDMD, cell viability, and LDH activity in chondrocytes were assessed.
View Article and Find Full Text PDFlinc00511 Knockdown Inhibits Lung Cancer Progression by Regulating miR-16-5p/MMP11.
Crit Rev Eukaryot Gene Expr
August 2023
Department of Oncology, Xiangyang No. 1 People's Hospital, Hubei University of Medicine, Xiangyang 441000, Hubei, China.
Lung cancer (LC) is a malignant tumor that extremely impairs people. According to numerous studies, long non-coding RNA (lncRNA) was inextricably involved in the advancement of LC. The work aspired to identify linc00511 expression in LC and to dig for the underlying mechanisms linc00511 regulated LC progression.
View Article and Find Full Text PDFLINC00511 promotes melanoma progression by targeting miR-610/NUCB2.
Open Med (Wars)
March 2023
Department of Dermatology, Hebei Province Cangzhou Central Hospital, Hebei, 061001, China.
Long intergenic noncoding RNA 00511 (LINC00511) predicts poor prognosis in various malignancies and functions as an oncogene in distinct malignant tumors. The role of LINC00511 in melanoma progression was assessed. In our research, expression of LINC00511 in melanoma cells was detected by quantitative reverse transcription PCR.
View Article and Find Full Text PDFLinc00511 Knockdown Inhibited TGF-β1-Induced Epithelial-Mesenchymal Transition of Bronchial Epithelial Cells by Targeting miR-16-5p/Smad3.
Am J Rhinol Allergy
May 2023
Department of Respiratory and Critical Care Medicine, 477248Nanxishan Hospital of Guangxi Zhuang Autonomous Region, Guilin, China.
Background: Airway remodeling in patients with asthma was correlated with induced epithelialmesenchymal transition (EMT) of bronchial epithelial cells.
Objective: This study examined the mechanism of Linc00511 on induced EMT of bronchial epithelial cells after transforming growth factor-β1 (TGF-β1) induction.
Methods: The human bronchial epithelial cell 16HBE was treated with 10 ng/mL TGF-β1 for 12 h, 24 h, or 48 h to induce EMT.
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