AI Article Synopsis

  • The study highlights the use of machine-learning algorithms to identify genetic disorders by analyzing facial photographs, revealing potential applications beyond known phenotypes.
  • Researchers identified two individuals with a novel genetic disorder linked to a specific mutation in the LEMD2 gene, displaying similar progeria-like facial features and unique physical and neurological anomalies.
  • The findings suggest that artificial intelligence can aid in discovering new genetic disorders by clustering similar syndromes, indicating a common genetic cause, while also noting that the prognosis for this condition is better than classic Hutchinson-Gilford progeria.

Article Abstract

Over a relatively short period of time, the clinical geneticist's "toolbox" has been expanded by machine-learning algorithms for image analysis, which can be applied to the task of syndrome identification on the basis of facial photographs, but these technologies harbor potential beyond the recognition of established phenotypes. Here, we comprehensively characterized two individuals with a hitherto unknown genetic disorder caused by the same de novo mutation in LEMD2 (c.1436C>T;p.Ser479Phe), the gene which encodes the nuclear envelope protein LEM domain-containing protein 2 (LEMD2). Despite different ages and ethnic backgrounds, both individuals share a progeria-like facial phenotype and a distinct combination of physical and neurologic anomalies, such as growth retardation; hypoplastic jaws crowded with multiple supernumerary, yet unerupted, teeth; and cerebellar intention tremor. Immunofluorescence analyses of patient fibroblasts revealed mutation-induced disturbance of nuclear architecture, recapitulating previously published data in LEMD2-deficient cell lines, and additional experiments suggested mislocalization of mutant LEMD2 protein within the nuclear lamina. Computational analysis of facial features with two different deep neural networks showed phenotypic proximity to other nuclear envelopathies. One of the algorithms, when trained to recognize syndromic similarity (rather than specific syndromes) in an unsupervised approach, clustered both individuals closely together, providing hypothesis-free hints for a common genetic etiology. We show that a recurrent de novo mutation in LEMD2 causes a nuclear envelopathy whose prognosis in adolescence is relatively good in comparison to that of classical Hutchinson-Gilford progeria syndrome, and we suggest that the application of artificial intelligence to the analysis of patient images can facilitate the discovery of new genetic disorders.

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC6451726PMC
http://dx.doi.org/10.1016/j.ajhg.2019.02.021DOI Listing

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