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Warm autoimmune hemolytic anemia with anti-Jk specificity following babesiosis masquerading as a delayed hemolytic transfusion reaction.
Transfusion
April 2023
Department of Laboratory Medicine, Yale School of Medicine, New Haven, Connecticut, USA.
Background: Warm autoimmune hemolytic anemia (WAIHA) is characterized by the development of autoantibodies that react with red blood cells (RBCs) optimally at physiologic temperature, classically resulting in a positive direct antiglobulin test (DAT) for IgG and a panreactive eluate. Babesiosis has been described as a potentiator of WAIHA, and all cases have shown classic blood bank findings. Only rare reports have described autoantibodies, both secondary to babesiosis and overall, with specificity for Kidd antigens.
View Article and Find Full Text PDFSevere Babesiosis Masquerading as Thrombotic Thrombocytopenic Purpura: A Case Report.
Cureus
April 2019
Nephrology, New York-Presbyterian Queens, Flushing, USA.
Babesiosis is an infectious disease caused by protozoa of the genus Babesia. Its incidence has been increasing in the Northeast and upper Midwest regions of the United States in recent years. Symptoms of babesiosis can range from a mild flu-like illness to acute, severe, and fatal disease.
View Article and Find Full Text PDFBabesiosis Masquerading as Evans Syndrome.
Am J Med
July 2019
Department of Medicine, University of Maryland School of Medicine, Baltimore, Md; University of Maryland Greenebaum Comprehensive Cancer Center, Baltimore, Md.
Low-virulent Babesia venatorum infection masquerading as hemophagocytic syndrome.
Ann Hematol
April 2018
Department of Laboratory Medicine Lund, Lund University, Skåne University Hospital, Lund, Sweden.
Hepatosplenic alpha/beta T-cell lymphoma masquerading as cirrhosis.
J Gastrointest Oncol
June 2013
Division of Gastroenterology, Department of Medicine, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts, USA;
A 59-year-old man with diabetes mellitus, prior hepatitis B infection and recently diagnosed cirrhosis with prior Babesiosis presented to our institution from an outside hospital with six months of worsening abdominal pain, myalgias and fevers. On admission, physical examination revealed jaundice, hepatosplenomegaly and diffuse lymphadenopathy. Laboratory investigations demonstrated mild anemia, thrombocytopenia, hyperbilirubinemia and elevated lactate dehydrogenase.
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