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Ambient Air Pollution and Congenital Heart Disease: Updated Evidence and Future Challenges.
Antioxidants (Basel)
January 2025
Institute of Clinical Physiology, National Research Council, 56124 Pisa, Italy.
Congenital heart disease (CHD) represents the major cause of infant mortality related to congenital anomalies globally. The etiology of CHD is mostly multifactorial, with environmental determinants, including maternal exposure to ambient air pollutants, assumed to contribute to CHD development. While particulate matter (PM) is responsible for millions of premature deaths every year, overall ambient air pollutants (PM, nitrogen and sulfur dioxide, ozone, and carbon monoxide) are known to increase the risk of adverse pregnancy outcomes.
View Article and Find Full Text PDFAssociation between joint exposure to ambient air pollutants and carotid plaque: The mediating role of cardiometabolic risk factors.
Ecotoxicol Environ Saf
January 2025
Beijing Municipal Key Laboratory of Clinical Epidemiology, Department of Epidemiology and Health Statistics, School of Public Health, Capital Medical University, Beijing, China; Harvard T.H. Chan School of Public Health, 655 Huntington Ave, Boston, MA 02115, USA. Electronic address:
Background: Research has shown that exposure to joint air pollution is related to atherosclerosis, but little evidence has been found for carotid plaques. Our objective is to assess the association between exposure to joint air pollutants and carotid plaque and explore the mediating role of cardiometabolic factors in this relationship.
Methods: The Beijing Health Management Cohort (BMHC) study followed participants recruited from 2013 to 2014 until December 31, 2020.
Short-Term Effect of Air Pollution on Daily Hospital Visits for Anxiety Disorders in Southern China with Low Pollution Concentrations.
Toxics
January 2025
Department of Medical Statistics, School of Public Health, Sun Yat-sen University, Guangzhou 510080, China.
The global prevalence and burden of anxiety disorders (ADs) are increasing. However, findings on the acute effects of air pollution on ADs remain inconclusive. We evaluated the effects of short-term exposure to ambient air pollutants, including fine particulate matter (PM), inhalable particulate matter (PM), nitrogen dioxide (NO), carbon monoxide (CO), sulfur dioxide (SO), and ozone (O), on daily hospital visits for ADs.
View Article and Find Full Text PDFAmbient Air Pollution and COPD: The Multiethnic Cohort Study.
Ann Am Thorac Soc
January 2025
University of California San Francisco, Department of Epidemiology and Biostatistics, San Francisco, California, United States.
Rationale: Globally, in 2019, chronic obstructive pulmonary disease (COPD) was the third leading cause of death. While tobacco smoking is the predominant risk factor, the role of long-term air pollution exposure in increasing risk of COPD remains unclear. Moreover, there are few studies that have been conducted in racial and ethnic minoritized and socioeconomically diverse populations, while accounting for smoking history and other known risk factors.
View Article and Find Full Text PDFAssociations of Short-Term Ozone Exposure With Hypoxia and Arterial Stiffness.
J Am Coll Cardiol
January 2025
SKL-ESPC & SEPKL-AERM, College of Environmental Sciences and Engineering, Center for Environment and Health, Peking University, Beijing, China; Research Station of Alpine Ecology Environment and Health at Tibet University, Lhasa, Tibet Autonomous Region, China. Electronic address:
Background: Epidemiological studies reported associations between ozone (O) exposure and cardiovascular diseases, yet the biological mechanisms remain underexplored. Hypoxia is a shared pathogenesis of O-associated diseases; therefore, we hypothesized that O exposure may induce changes in hypoxia-related markers, leading to adverse cardiovascular effects.
Objectives: This study aimed to investigate associations of short-term O exposure with hypoxic biomarkers and arterial stiffness.
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