A case of primary aldosteronism who experienced cardiopulmonary arrest, was resuscitated and cured.

A 45-year-old female went into cardiopulmonary arrest. She was in ventricular fibrillation (VF) and was defibrillated using an automated external defibrillator. After arrival at our hospital, electrocardiography monitoring showed QT prolongation. Serum potassium was low at 2.2 mEq/L, and hypokalemia-induced long QT syndrome was considered to be the cause of this patient's VF. An intravenous infusion of potassium and magnesium sulphate was started, which normalized her serum potassium and QTc interval, with no recurrence of ventricular arrhythmias. Endocrinological investigations showed a plasma renin activity of <0.1 ng/(mL h) and a plasma aldosterone concentration 258 pg/mL. Computed tomography scanning revealed a low signal area 16 mm × 20 mm in size of the right adrenal gland. From the above findings, this patient was diagnosed with a right adrenal tumor and primary aldosteronism. We concluded that the right adrenal tumor was excreting excess amounts of aldosterone from adrenal vein sampling, and performed laparascopic right adrenalectomy. Serum potassium levels rose immediately to normal levels postoperatively. We were able to withdraw her antihypertensive medication 3 months after adrenalectomy. We report a case of primary aldosteronism who experienced cardiopulmonary arrest, was resuscitated, and cured. < When you come across ventricular fibrillation, please consider one of the reasons is caused by hypokalemia due to primary aldosteronism. After an appropriate resuscitation, both hypokalemia and hypertension are completely curable by removing the adrenal tumor.>.