A 23-year-old man was referred to our center with hematuria and hemolysis. The patient had undergone mitral and tricuspid valve replacement 3 months previously. Echocardiography and catheterization revealed a Gerbode-type ventricular septal defect. A decision was made to occlude the defect interventionally. The patient's hematuria ceased immediately after the occlusion of the defect. < Iatrogenic ventricular septal defects (especially Gerbode-type) are relatively rare complications after valvular surgery. Correction of such defects can be done both surgically and interventionally, but since the risk of another operation for correction is high, percutaneous ventricular septal defect closure is usually the preferred treatment option. Using an appropriate approach will increase the success rate.>.
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http://dx.doi.org/10.1016/j.jccase.2015.04.006 | DOI Listing |
Kardiol Pol
January 2025
Department of Pediatric Cardiology, Shandong Provincial Hospital, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, China.
Am J Cardiol
December 2024
Inova Center of Outcomes Research, Inova Heart and Vascular, Fairfax, VA. Electronic address:
Cardiovasc Ther
January 2025
Department of Pediatric Cardiology Saarland University Medical Center, Homburg 66421, Germany.
The objective of this study is to evaluate the clinical application and primary outcome of transcatheter embolization using Amplatzer™ Vascular Plug (AVP) Type 2 and Type 4 in different congenital cardiovascular malformations. This is a single-center retrospective observational cohort study. We analyzed clinical and imaging data of 36 patients retrospectively who received transcatheter embolizations of the following malformations using AVP: systemic-to-pulmonary collateral arteries (SPCA), patent ductus arteriosus (PDA), ventricular septal defects (VSD), and aberrant pulmonary sequestration arteries (PSA).
View Article and Find Full Text PDFEur Heart J Imaging Methods Pract
July 2024
Department of Nuclear Medicine, CHU de Caen Normandie, Normandie Univ, UNICAEN UR 4650 PSIR, Avenue Cote de Nacre, 14000 Caen, France.
Front Cardiovasc Med
December 2024
Department of Cardiology, Medical University of Vienna, Vienna, Austria.
Introduction: Preeclampsia (PE) is thought to be the consequence of impaired placental perfusion leading to placental hypoxia. While it has been demonstrated that PE may be a consequence of maternal cardiovascular maladaptation, the exact role of maternal cardiac function remains to be determined. This study sought to assess cardiac characteristics in pregnant women diagnosed with PE and to determine the possible relationship between PE, maternal cardiac changes/function, and NT-proBNP levels.
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