Societal impact is an increasingly important imperative of academic funding. However, there is little research to date documenting how impact is accomplished in practice. Drawing on insights from Actor-Network Theory, we explore the research-policy interface within an interdisciplinary research project on the relationships between air pollution and human health. Health policy impact was important to the researchers for moral as well as pragmatic reasons but it was a goal that was seen as potentially in tension with that of doing science. In fields such as air pollution and health, networks of policymakers and researchers are inevitably entangled, and we found that processes of engagement operated to delineate science from policy. Health was initially black-boxed and under-explicated, used as a signifier in itself for societal impact. By mobilising networks of policy actors, brought together in workshops to rank the importance of policy scenarios for the research team, the connections between air pollution and health were materialised and made actionable. This was achieved by framing existing data sets, emission technologies, policy expertise, pollutant species and human health in particular ways and, in doing so, excluding others. The process of linking air pollution and health research to achieve societal impact not only influenced how these phenomena were known but, critically, enabled and constrained potential policy responses. Tracing these research arrangements made the material discursive processes of 'impact' visible and analysable as objects of social science scholarship, and therefore generated a productive site for critically engaging with processes of environment and health science and policy.
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http://dx.doi.org/10.1177/1363459318804590 | DOI Listing |
Part Fibre Toxicol
March 2025
IUF - Leibniz Research Institute for Environmental Medicine, Auf'm Hennekamp 50, 40225, Düsseldorf, Germany.
Background: Inhalation of combustion-derived nanoparticles may contribute to the development or exacerbation of inflammatory lung diseases by direct interaction with neutrophilic granulocytes. Earlier studies have shown that exposure of human neutrophils to carbon nanoparticles ex vivo causes a prolongation of cellular life by the reduction of apoptosis rates. Accordingly, reduced neutrophil apoptosis rates were observed in neutrophils from bronchoalveolar lavages from carbon nanoparticle-exposed animals.
View Article and Find Full Text PDFJ Asthma
March 2025
Department of Epidemiology and Biostatistics, College of Public Health, Zhengzhou University, Zhengzhou, Henan, PR China.
J Environ Manage
March 2025
European Commission, Joint Research Centre (JRC), Ispra, Italy.
PLoS One
March 2025
Department of Occupational Environmental Medicine, Seoul Medical Center, Seoul, Korea.
Risk factors for asthma include genetic, host, and environmental factors such as allergens, smoking, and exposure to chemicals. Heavy metals from air pollution or contaminated water and food can also trigger asthma. This study aimed to identify the biological exposure levels of blood lead, mercury, and cadmium, and determine the association of asthma with single and multiple exposures to these heavy metals using data from the Korean National Health and Nutrition Examination Survey (KNHANES) conducted between 2008 and 2013.
View Article and Find Full Text PDFPLoS One
March 2025
School of Public Health, University of California, Berkeley, California, United States of America.
Background: Hypertension is a leading risk factor for cardiovascular disease, and its association with household air pollution (HAP) in sub-Saharan Africa is understudied.
Main Objective: To investigate the association between blood pressure (BP) and HAP exposure in a population-based cohort in rural Malawi.
Materials And Methods: In the Chikwawa district, the site of a previous randomized controlled trial of a cleaner-burning cookstove intervention (the Cooking and Pneumonia Study or CAPS), we recruited 1,481 randomly selected adults.
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