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http://dx.doi.org/10.1016/j.radcr.2018.04.003 | DOI Listing |
JAMA Neurol
January 2025
Senior Department of Neurosurgery, the First Medical Center of PLA General Hospital, Hai-dian District, Beijing, China.
Interv Neuroradiol
December 2024
Department of Neurology, Mayo Clinic, Rochester, MN, USA.
Background: Iatrogenic cervical artery dissection (CeAD) results from various procedures including interventional angiographic procedures and diagnostic angiography. Iatrogenic CeAD is rare, resulting in limited literature on management and outcomes. This observational cohort study investigates approaches and outcomes of iatrogenic CeAD after endovascular interventions.
View Article and Find Full Text PDFAm J Ophthalmol Case Rep
December 2024
Kaiser Permanente Northern California, Department of Ophthalmology, Oakland, CA, USA.
Purpose: To describe the visual acuity (VA) outcomes from a telemedicine-enabled pathway allowing for rapid diagnosis and administration of intravenous (IV) thrombolytic treatment for non-arteritic central retinal artery occlusion (naCRAO) within 4.5 hours (4.5 h) of visual loss.
View Article and Find Full Text PDFEur Stroke J
October 2024
Department of Neurology, Charité - Universitätsmedizin Berlin, Corporate member of Freie Universität Berlin and Humboldt Universität zu Berlin, Berlin, Germany.
Background: Acute intracranial occlusion of the internal carotid artery (ICA) can be distinguished into (a) occlusion of the terminal ICA, involving the proximal segments of the middle or anterior cerebral artery (ICA-L/-T) and (b) non-terminal intracranial occlusions of the ICA with patent circle of Willis (ICA-I). While patients with ICA-L/-T occlusion were included in all randomized controlled trials on endovascular therapy (EVT) in anterior large vessel occlusion, data on EVT in ICA-I occlusion is scarce. We thus aimed to evaluate effectiveness and safety of EVT in ICA-I occlusions in comparison to ICA-L/-T occlusions.
View Article and Find Full Text PDFBiomedicines
September 2024
Department of Neurology, University Hospital Würzburg, 97080 Würzburg, Germany.
Severe acute ischemic stroke (AIS) is mainly caused by thromboembolism originating from symptomatic carotid artery (ICA) stenosis or in the heart due to atrial fibrillation. Glycoprotein VI (GPVI), a principal platelet receptor, facilitates platelet adherence and thrombus formation at sites of vascular injury such as symptomatic ICA stenosis. The shedding of GPVI from the platelet surface releases soluble GPVI (sGPVI) into the circulation.
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