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http://dx.doi.org/10.1016/j.jacep.2016.12.012 | DOI Listing |
We illustrate the case Brugada Type 1 pattern on electrocardiogram in a setting of hyperkalemia, changes which were reversible following normalization of serum potassium levels. Although Brugada Type 1 syndrome is associated with sudden cardiac death, a quick search for alternate reversible pathology is essential to timely management and avoid unnecessary cardiac intervention.
View Article and Find Full Text PDFJACC Clin Electrophysiol
September 2017
Cardiovascular Division, Department of Medicine, University of Minnesota, Minneapolis, Minnesota; Cardiac Arrhythmia and Syncope Center, Cardiovascular Division, University of Minnesota Medical Center, Minneapolis, Minnesota. Electronic address:
Am J Cardiol
March 2018
Department of Medicine, Queen's University, Kingston, Ontario, Canada. Electronic address:
Brugada phenocopies (BrPs) are clinical entities that differ in etiology from true congenital Brugada syndrome but have identical electrocardiographic (ECG) patterns. Hyperkalemia is known to be one of the causes of BrP. The aim of this study was to determine the clinical characteristics and evolution of hyperkalemia-induced BrP.
View Article and Find Full Text PDFCase Rep Cardiol
February 2017
Lehigh Valley Hospital, Department of Cardiovascular Medicine, 1627 W. Chew St., Allentown, PA 18104, USA.
Brugada syndrome (BrS) is an inherited disorder of cardiac ion channels characterized by peculiar ECG findings predisposing individuals to ventricular arrhythmias, syncope, and sudden cardiac death (SCD). Various electrolyte disturbances and ion channels blocking drugs could also provoke BrS ECG findings without genetic BrS. Clinical differentiation and recognition are essential for guiding the legitimate action.
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