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| http://dx.doi.org/10.3389/fped.2018.00099 | DOI Listing |
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Confronting the "Weaponization" of Genetics by Racists Online and Elsewhere.
Hastings Cent Rep
December 2024
Genomics research is regularly appropriated in social and political contexts to publicly legitimize unjust and malicious political views, policies, and actions. In recent years, there have been high-profile cases of mass shooters, public intellectuals, and political insiders using genomics findings to convince audiences that deadly force and coercive policies against racial minorities are warranted. To create a just genomics, geneticists must consider what makes their research so attractive and adaptable for the legitimization of unjust ends and what they can do to counter such appropriations.
View Article and Find Full Text PDFComputational studies on metabolic pathways of Coxiella burnetii to combat Q fever: A roadmap to vaccine development.
Microb Pathog
January 2025
Department of Animal Sciences, School of Life Sciences, Central University of Himachal Pradesh, District Kangra, Himachal Pradesh, India, 176206. Electronic address:
Coxiella burnetii (Cbu) is the gram-negative intracellular pathogen responsible for deadly zoonotic infection, Q fever. The pathogen is environmentally stable and distributed throughout the world which is sustained in nature by chronic infection of ruminants. The epidemiological studies on Q fever indicates it as emerging public health problem in various countries and it is imperative to promptly identify an appropriate therapeutic solution for this pathogen.
View Article and Find Full Text PDFRevisiting Vitamin D Guidelines: A Critical Appraisal of the Literature.
Endocr Pract
December 2024
Section of Endocrinology, Diabetes, Nutrition and Weight Management, Departments of Medicine, Pharmacology, Physiology and Biophysics and Molecular Medicine, Boston University School of Medicine, Boston, Massachusetts. Electronic address:
Article Synopsis
- The review compares the 2024 and 2011 Endocrine Society's Clinical Practice Guidelines on vitamin D, highlighting a shift from managing deficiency in the 2011 guidelines to broader health benefits for the general population in the 2024 guidelines.
- In 2024, recommendations are made for specific groups, such as children, pregnant women, and those over 75, while the 2011 guidelines focused on treating existing deficiencies and their complications.
- The 2024 guidelines recommend taking 600-800 IUs of vitamin D for nonpregnant adults up to 75 years old, but caution that these do not apply to individuals with certain metabolic abnormalities, diverging from the 2011 guidelines which emphasized maintaining higher vitamin D
Phosphorylation in the Proteome: A Meta-Analysis of Publicly Available Data Sets.
J Proteome Res
December 2024
Institute of Systems, Molecular and Integrative Biology, University of Liverpool, Liverpool L69 7BE, United Kingdom.
Malaria is a deadly disease caused by Apicomplexan parasites of the genus. Several species of the genus are known to be infectious to humans, of which is the most virulent. Post-translational modifications (PTMs) of proteins coordinate cell signaling and hence regulate many biological processes in homeostasis and host infection, of which the most highly studied is phosphorylation.
View Article and Find Full Text PDFRole of the SaeRS Two-Component Regulatory System in Group B Biofilm Formation on Human Fibrinogen.
Microorganisms
October 2024
Department of Human Pathology of Adult and Developmental Age "Gaetano Barresi", University of Messina, 98168 Messina, Italy.
, also known as Group B or GBS, is a commensal colonizer of human vaginal and gastrointestinal tracts that can also be a deadly pathogen for newborns, pregnant women, and the elderly. The SaeRS two-component regulatory system (TCS) positively regulates the expression of two GBS adhesins genes, but its role in the formation of biofilm, an important step in pathogenesis, has not been investigated. In the present study, we set up a novel model of GBS biofilm formation using surfaces coated with human fibrinogen (hFg).
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