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Visceral leishmania-associated hemophagocytic lymphohistiocytosis.
Med Clin (Barc)
September 2018
Servicio de Neurología, Hospital Universitario Vall d'Hebron, Barcelona, España.
Distinct cellular migration induced by Leishmania infantum chagasi and saliva from Lutzomyia longipalpis in a hemorrhagic pool model.
Rev Inst Med Trop Sao Paulo
April 2014
Faculdade de Medicina, Universidade Federal do Ceará, 60430-160FortalezaCE, Brazil, Faculdade de Medicina, Universidade Federal do Ceará, Rua Alexandre Baraúna 949, 60430-160 Fortaleza, CE, Brazil.
Recruitment of a specific cell population after Leishmania infection can influence the outcome of the disease. Cellular migration in response to Leishmania or vector saliva has been reported in air pouch model, however, cellular migration induced by Leishmania associated with host's blood and vector saliva in this model has not been described. Herein we investigated cellular migration into air pouch of hamster after stimulation with combination of L.
View Article and Find Full Text PDFAdenosine and immune imbalance in visceral leishmaniasis: the possible role of ectonucleotidases.
J Trop Med
November 2011
Instituto de Bioquímica Médica, Universidade Federal do Rio de Janeiro, CCS, Bloco H, Cidade Universitária, Ilha do Fundão, 21941-590 Rio de Janeiro, RJ, Brazil.
Visceral leishmaniasis (VL) is the most severe form of leishmaniasis and is responsible for most Leishmania-associated deaths. VL represents a serious public health problem that affects many countries. The immune response in leishmaniasis is very complex and is poorly understood.
View Article and Find Full Text PDFHemophagocytic lymphohistiocytosis associated with visceral leishmaniasis: a case report.
Pediatr Hematol Oncol
November 2005
Department of Pediatrics, Başkent University, Faculty of Medicine, Adana Teaching and Medical Research Center, Adana, Turkey.
Leishmania-associated hemophagocytic lymphohistiocytosis is a rare clinicopathological entity. This condition is often difficult to diagnose, so treatment is often delayed. This report describes the case of a 5-year-old boy who was admitted with fever of 1 month's duration, hepatosplenomegaly, and pancytopenia.
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