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| http://dx.doi.org/10.1073/pnas.1805691115 | DOI Listing |
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Lyme disease and relapsing fever Borrelia elongate through zones of peptidoglycan synthesis that mark division sites of daughter cells.
Proc Natl Acad Sci U S A
August 2016
Microbial Sciences Institute, Yale University, West Haven, CT 06517; Department of Molecular, Cellular, and Developmental Biology, Yale University, New Haven, CT 06516; Howard Hughes Medical Institute, Yale University, New Haven, CT 06516; Department of Microbial Pathogenesis, Yale Medical School, New Haven, CT 06516
Agents that cause Lyme disease, relapsing fever, leptospirosis, and syphilis belong to the phylum Spirochaetae-a unique lineage of bacteria most known for their long, spiral morphology. Despite the relevance to human health, little is known about the most fundamental aspects of spirochete growth. Here, using quantitative microscopy to track peptidoglycan cell-wall synthesis, we found that the Lyme disease spirochete Borrelia burgdorferi displays a complex pattern of growth.
View Article and Find Full Text PDFShigella impairs T lymphocyte dynamics in vivo.
Proc Natl Acad Sci U S A
March 2013
Molecular Microbial Pathogenesis Unit, Department Cell Biology and Infection, Institut National de Santé et de Recherche Médicale U786, 75724 Paris Cedex 15, France.
The Gram-negative enteroinvasive bacterium Shigella flexneri is responsible for the endemic form of bacillary dysentery, an acute rectocolitis in humans. S. flexneri uses a type III secretion system to inject effector proteins into host cells, thus diverting cellular functions to its own benefit.
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