[This corrects the article DOI: 10.18632/oncotarget.20687.].
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http://dx.doi.org/10.18632/oncotarget.24848 | DOI Listing |
Cell Death Dis
November 2024
Key Laboratory of Carcinogenesis and Cancer Invasion, Liver Cancer Institute, Zhongshan Hospital, Fudan University, Ministry of Education, Shanghai, 200032, China.
[This corrects the article DOI: 10.1371/journal.pone.
View Article and Find Full Text PDFOncol Rep
June 2024
Department of Orthopaedics, The First Affiliated Hospital of Harbin Medical University, Harbin, Heilongjiang 150001, P.R. China.
Following the publication of the above article, an interested reader drew to the authors' attention that, for the cell invasion assay experiments shown in Fig. 2D on p. 5, there appeared to be an overlapping section of data comparing between the Sao‑2/Control and MG‑63/siH19 panels, such that these data had been derived from the same original source where the panels were intended to portray the results from differently performed epxeriments.
View Article and Find Full Text PDFEur J Pharmacol
November 2022
Chang Gung University College of Medicine, Kaohsiung, 83301, Taiwan; Kawasaki Disease Center, Kaohsiung Chang Gung Memorial Hospital, Kaohsiung, 83301, Taiwan; Department of Pediatrics, Kaohsiung Chang Gung Memorial Hospital, Kaohsiung, 83301, Taiwan. Electronic address:
Parkinson's disease (PD) primarily affects the motor system and is the second most common age-related neurodegenerative disorder after Alzheimer's disease. Mitochondrial complex I deficiency and functional abnormalities are implicated in the development of PD. MicroRNA-29a (miR-29a) has emerged as a critical miRNA in PD.
View Article and Find Full Text PDFGene Ther
December 2019
Department of Neuroscience, College of Medicine University of Florida, Gainesville, FL, 32610, USA.
The majority of hereditary neuropathies are caused by duplication of the peripheral myelin protein 22 (PMP22) gene. Therefore, mechanisms to suppress the expression of the PMP22 gene have high therapeutic significance. Here we asked whether the human PMP22 gene is a target for regulation by microRNA 29a (miR-29a).
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