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| http://dx.doi.org/10.1212/CPJ.0000000000000156 | DOI Listing |
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Erythropoietin-Resistant Anemia Secondary to Zinc-Induced Hypocupremia in a Hemodialysis Patient.
Case Rep Nephrol Dial
July 2021
East Georgia Kidney and Hypertension, Augusta, Georgia, USA.
Excessive intake of zinc is a known but often forgotten cause of copper deficiency, and its consequences in the context of end-stage renal disease (ESRD) are not widely discussed. Zinc-induced copper deficiency (ZICD) can result in erythropoietin (EPO)-resistant anemia and may not be considered as a possible etiology when conducting the work-up. We present a case wherein an ESRD patient had been receiving excess zinc for several months and subsequently experienced EPO-resistant anemia.
View Article and Find Full Text PDFHypocupraemia-induced anaemia, sensory ataxia and cognitive impairment secondary to zinc-containing dental adhesive.
BMJ Case Rep
July 2021
Neurology, University Hospital Southampton NHS Foundation Trust, Southampton, Hampshire, UK.
A 67-year-old man presented with 5 months of worsening memory impairment and sensory gait ataxia on the background of symptomatic anaemia. He experienced falls, agitation and became socially withdrawn over 3 weeks, resulting in hospital admission. On examination, he had sensory gait ataxia consistent with a dorsal column syndrome.
View Article and Find Full Text PDFA Unique Case of Severe Anemia Secondary to Copper Deficiency in an Adult Patient.
Cureus
November 2018
Internal Medicine, University of Arkansas for Medical Sciences, Little Rock, USA.
Anemia is a frequently encountered problem in the healthcare system. Common causes of anemia include blood loss, followed by impaired red blood cell production and red blood cell destruction. This case demonstrates the need for cognizance of the less frequent causes of anemia.
View Article and Find Full Text PDFZinc containing dental fixative causing copper deficiency myelopathy.
BMJ Case Rep
August 2017
Department of Neurology, Institute of Neurological Sciences, Queen Elizabeth University Hospital, Glasgow, UK.
A 62-year-old male, previously well, was referred to neurology clinic following 6 months history of worsening lower limbs instability, paraesthesia, pain and weakness rendering him housebound. Examination revealed upper motor neuron pattern of weakness of the lower limbs and loss of proprioception. Serum analysis revealed reduced caeruloplasmin and copper levels with raised zinc.
View Article and Find Full Text PDFSubacute copper-deficiency myelopathy in a patient with occult celiac disease.
J Spinal Cord Med
July 2017
a Department of Neuroscience , S. Agostino-Estense Hospital and University of Modena and Reggio Emilia, Via Pietro Giardini 1355 IT-41126 Modena , Italy.
Context: Acquired copper deficiency represents a rare cause of progressive myelopathy presenting with sensory ataxia and spastic gait. The time interval from neurological symptoms onset to diagnosis of myelopathy ranges from 2 months to several years in almost all cases, mimicking the clinical course of subacute combined degeneration due to vitamin B12 deficiency.
Findings: A 60-year-old man, without any gastrointestinal symptoms, developed over the course of one week rapidly progressive gait imbalance, tingling and numbness in his feet and ascending lower limb weakness.
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