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Bubbles in the Heart: A Case Series of Three Varying Presentations of Hepatopulmonary Syndrome.
J Pharm Bioallied Sci
December 2024
Department of General Medicine, Pushpagiri Institute of Medical Sciences and Research Centre, Thiruvalla, Kerala, India.
Hepatopulmonary syndrome (HPS) is described as an arterial oxygenation deficit caused by intrapulmonary vascular dilatations in the presence of chronic liver disease. This case series highlighted three HPS cases that were brought to the emergency unit of the institution. This helps clinicians evaluate and treat hepatopulmonary syndrome and emphasizes the involvement of interprofessional teamwork.
View Article and Find Full Text PDFSurvival of patients with hepatopulmonary syndrome related to cirrhotic and non-cirrhotic (schistosomiasis) portal hypertension.
World J Hepatol
February 2025
Postgraduate Program in Tropical Medicine, Center of Medical Sciences, Universidade Federal de Pernambuco, Recife 50670901, Pernambuco, Brazil.
Background: The hepatosplenic schistosomiasis (HSS) with portal hypertension can cause vascular complications such as hepatopulmonary syndrome (HPS). HPS increases the risk of mortality in patients with cirrhosis; however, there is no data on the mortality of patients with HSS and HPS.
Aim: To perform a survival analysis of patients with HPS related to cirrhotic and non-cirrhotic (schistosomiasis) portal hypertension.
Placental growth factor modulates endothelial NO production and exacerbates experimental hepatopulmonary syndrome.
JHEP Rep
March 2025
Université Paris-Saclay, Unité Mixte de Recherche en Santé (UMR_S) 999 Hypertension Pulmonaire: Physiopathologie et Innovation Thérapeutique (HPPIT), Le Kremlin-Bicêtre, France.
Background & Aims: Hepatopulmonary syndrome (HPS) results from portal hypertension, with or without cirrhosis, and is marked by pulmonary vascular dilations leading to severe hypoxemia. Although placental growth factor (PlGF) is important for vascular growth and endothelial function, its role in HPS is unclear. This study investigated the involvement of PlGF in experimental models of HPS and in patients.
View Article and Find Full Text PDFInhaled nitric oxide as a salvage therapy for refractory hypoxemia in the post-transplantation period of hepatopulmonary syndrome: An explorative report of three cases.
Liver Res
September 2024
Surgical Intensive Care Unit, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, Guangdong, China.
Liver transplantation (LT) is the only effective treatment for hepatopulmonary syndrome (HPS). Moreover, perioperative refractory hypoxemia (pRH) is a prevalent life-threatening condition and has extremely limited treatment options. Here, we report three patients with HPS who experienced pRH after LT and were consecutively treated with different salvage therapies, ephedrine inhalation, intravenous use of methylene blue with nitric oxide (NO) inhalation, and NO inhalation alone.
View Article and Find Full Text PDFModulating respiratory mechanics and inflammation in hepatopulmonary syndrome: Aerobic exercise as a therapeutic strategy.
Respir Physiol Neurobiol
February 2025
Laboratory of Respiratory Physiology, University of Brasilia, Brasília, DF, Brazil; Graduate Program in Medical Sciences, School of Medicine, University of Brasilia, Brasília, DF, Brazil; Division of Pulmonology, Brasilia University Hospital, Brasília, DF, Brazil.
Introduction: Aerobic exercise training positively modulates the immune system and improves lung function; however, its effects on respiratory system's elastic, resistive properties and interleukin-10 (IL-10) concentration in hepatopulmonary syndrome (HPS) remains unexplored. This study aimed to assess whether moderate-intensity aerobic (AE) training altered exercise capacity, respiratory mechanics and lung inflammation.
Material And Methods: Wistar rats were randomly assigned to SHAM, HPS, HPS + AE4, and HPS + EA8 groups.
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