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Exercise capacity in a cohort of children with congenital heart disease.
Eur J Pediatr
January 2023
Department of Pediatrics, Division of Pediatric Cardiology, Erasmus MC, University Medical Center, Room number Sp2469 attn. Prof. Dr. W.A. Helbing, PO box 2040, 3000 CA, Zuid Holland, Rotterdam, The Netherlands.
In patients with congenital heart disease (CHD), reduced exercise capacity can be a predictor for late complications and may be used to guide interventions. Yet, the interpretation of exercise capacity is challenged by changes in body composition during growth. Our aim was to create an overview of disease-specific exercise capacity in children with CHD.
View Article and Find Full Text PDFCardiac Magnetic Resonance and Ventricular Arrhythmia Risk Assessment in Chronic Ischemic Cardiomyopathy: An Unmet Need?
Rev Cardiovasc Med
July 2022
Arrhytmia Section, Cardiology Department, Miguel Servet University Hospital, 50009 Zaragoza, Spain.
Ischemic cardiomyopathy (ICM) constitutes a major public health issue, directly involved in the prevalence and incidence of heart failure, ventricular arrhythmias (VA) and sudden cardiac death (SCD). Severe impairment of left ventricular ejection fraction (LVEF) is considered a high-risk marker for SCD, conditioning the criteria that determine an implantable cardiac defibrillator (ICD) placement in primary prevention according to current clinical guidelines. However, its sensitivity and specificity values for the prediction of SCD in ICM may not be highest.
View Article and Find Full Text PDFLate arrhytmias after repair of atrioventricular septal defect: Down's Syndrome is not the culprit.
Int J Cardiol
March 2018
Department of Pediatrics, Sapienza University of Rome, Rome, Italy, Viale Regina Elena, 324, Italy. Electronic address:
Objective: An overwiev of the new diagnostic method of fetal wellbeing - fetal magnetocardiography (fMCG).
Design: A review article.
Setting: Department of Gynecology and Obstetrics, Jessenius Faculty of Medicine in Martin, Comenius University in Bratislava, Slovak Republic.
Functional characterization of a novel frameshift mutation in the C-terminus of the Nav1.5 channel underlying a Brugada syndrome with variable expression in a Spanish family.
PLoS One
July 2014
Department of Pharmacology, School of Medicine, Universidad Complutense de Madrid, Madrid, Spain ; Instituto de Investigación Sanitaria Hospital Clínico San Carlos, School of Medicine, Universidad Complutense, Madrid, Spain.
Introduction: We functionally analyzed a frameshift mutation in the SCN5A gene encoding cardiac Na(+) channels (Nav1.5) found in a proband with repeated episodes of ventricular fibrillation who presented bradycardia and paroxysmal atrial fibrillation. Seven relatives also carry the mutation and showed a Brugada syndrome with an incomplete and variable expression.
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