[This corrects the article DOI: 10.1371/journal.pone.0030294.].

Download full-text PDF

Source
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5703075PMC
http://dx.doi.org/10.1371/annotation/1f110857-27d7-4e83-9eb3-4e5f51950a26DOI Listing

Publication Analysis

Top Keywords

correction wogonin
4
wogonin improves
4
improves histological
4
histological functional
4
functional outcomes
4
outcomes reduces
4
reduces activation
4
activation tlr4/nf-κb
4
tlr4/nf-κb signaling
4
signaling experimental
4

Similar Publications

Alzheimer's disease (AD) is a progressive neurodegenerative disease with memory loss and cognitive decline. Neurofibrillary tangles (NFTs) formed by hyperphosphorylated Tau protein are one of the pathological hallmarks of several neurodegenerative diseases including AD. Heat shock protein family B (small) member 1 (HSPB1) is a molecular chaperone that promotes the correct folding of other proteins in response to environmental stress.

View Article and Find Full Text PDF

Author Correction: Wogonin reversed resistant human myelogenous leukemia cells via inhibiting Nrf2 signaling by Stat3/ NF-κB inactivation.

Sci Rep

June 2021

State Key Laboratory of Natural Medicines, Jiangsu Key Laboratory of Carcinogenesis and Intervention, China Pharmaceutical University, 24 Tongjiaxiang, Nanjing, 210009, People's Republic of China.

View Article and Find Full Text PDF

Correction: Wogonin induces cell cycle arrest and erythroid differentiation in imatinib-resistant K562 cells and primary CML cells.

Oncotarget

January 2020

State Key Laboratory of Natural Medicines, Jiangsu Key Laboratory of Carcinogenesis and Intervention, Key Laboratory of Drug Quality Control and Pharmacovigilance, Ministry of Education, China Pharmaceutical University, 24 Tongjiaxiang, Nanjing, People's Republic of China.

[This corrects the article DOI: 10.18632/oncotarget.2340.

View Article and Find Full Text PDF

Want AI Summaries of new PubMed Abstracts delivered to your In-box?

Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!