Background: Stunting affects close to 2.4 million children in Uganda, and it is among the major contributors to the annual loss in productivity of the equivalent of US$899 million.
Objective: This study sought to identify the key underlying causes of the persistently high stunting rates in a region of Uganda known to have low poverty rates and a high agricultural output.
Methods: A matched case-control study was conducted in Buhweju district, southwest Uganda to compare prior exposures between stunted children (cases) and nonstunted children (controls).
Results: The sample comprised 56 cases and 112 controls, totaling 168 children aged 6 to 59 months. There were almost equal numbers of boys and girls in the sample. Boys were significantly more likely to be stunted compared to girls (odds ratio [OR]: 2.2, 95% confidence interval [CI]: 1.1-4.2; P < .05). Stunted children were significantly less likely to be introduced to complementary foods at an appropriate age and their primary caregivers lacked knowledge about stunting. The odds of stunting were 2.4 times greater for children in food-insecure households compared to those in food-secure households (OR: 2.4, 95% CI: 1.1-5.0; P < .05) and 4.5 times greater among children who resided in poor quality houses (OR: 4.5, 95% CI: 1.4-13.0; P < .05).
Conclusion: Low access to appropriate complementary diets, sex of child, food insecurity, poor socioeconomic status, and low knowledge about stunting were the main predictors of childhood stunting in the study population.
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http://dx.doi.org/10.1177/0379572117731666 | DOI Listing |
Am J Case Rep
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Observational studies have shown that the risk of developing herpes zoster (HZ) increases with the use of statins. However, there are many confounding factors in observational studies. Therefore, our Mendelian randomization (MR) study aimed to explore the causal role of lipids in HZ and to assess the causal impact of lipid-lowering drug targets on HZ risk.
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