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| http://dx.doi.org/10.3389/fped.2017.00167 | DOI Listing |
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Is the Prevalence of Biopsy-Proven Glomerulopathies in Adults Changing Over Time?
Maedica (Bucur)
September 2024
"Carol Davila" University of Medicine and Pharmacy, Bucharest, Romania.
Article Synopsis
- A study in Romania reviewed 1,254 adults with biopsy-proven glomerulopathies (GP) from 2008 to 2017 to assess changes in frequency and causes for kidney biopsies (KB).
- The research found that while nephrotic syndrome was a common reason for KB, its occurrence decreased significantly over the decade, with older patients showing more advanced kidney damage and chronic kidney function decline.
- Additionally, there was a notable increase in cases of diabetic nephropathy and crescentic glomerulonephritis, suggesting changes in diagnostic practices rather than an actual rise in GP prevalence among older adults.
Combination of Diuretics in States of Resistant Nephrotic Edema: a Case Presentation.
Maedica (Bucur)
September 2024
Department of Nephrology, "Carol Davila" University of Medicine and Pharmacy, Bucharest, Romania.
While there remains no universally accepted definition for resistant edema, it is generally acknowledged as edema that fails to respond to maximally administered doses of diuretics. Nephrotic edema is characterized by high levels of proteinuria, notably urinary concentrations of serine proteases, which possess the ability to activate the epithelial sodium channel (ENaC), resulting in persistent fluid retention. Loop diuretics are typically preferred as first-line therapy for hypervolemia.
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THSD7A-associated membranous nephropathy involves both complement-mediated and autonomous podocyte injury.
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Division of Nephrology, Department of Medicine, Toledo, OH, United States.
Membranous nephropathy (MN) continues to be a leading cause of nephrotic syndrome in non-diabetic adults. As a unique subtype in the serology-based classification of MN, thrombospondin type 1 domain containing 7A (THSD7A)-associated MN has attracted increasing interest, because, unlike other autoantigens, THSD7A is also expressed in preclinical species, facilitating the study of its role in MN. A heterologous mouse model of THSD7A-associated MN was previously established using a proprietary in-house antibody that was unfortunately not available to the research community.
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